Lobato Gustavo, Nakamura-Pereira Marcos
Department of Obstetrics, Fernandes Figueira Institute, Oswaldo Cruz Foundation (IFF-FIOCRUZ), Rio de Janeiro, Brazil.
Fetal Diagn Ther. 2008;24(4):474-7. doi: 10.1159/000178532. Epub 2008 Nov 29.
The Ballantyne syndrome (or mirror syndrome) is a gestational proteinuric hypertension associated with fetal hydrops. This report describes a case in which Ballantyne syndrome reversion occurred despite fetal hydrops persistence.
A 24-year-old woman showed fetoplacental hydrops at 28 2/7 gestational weeks. Severe Rh(D) alloimmunization and fetal hemolytic anemia (fetal hematocrit 15.4%) were confirmed by cordocentesis, and an intrauterine transfusion was performed. She also revealed hypertension (160/100 mm Hg), edema and proteinuria (845 mg/day). After four intrauterine transfusions, blood pressure was normalized; urinary proteinuria was not significant, and the edema vanished completely. Fetal hydrops persisted until delivery at 32 gestational weeks, but a partial reduction of placental hydrops was noted.
Total or partial reduction of the placental edema may be responsible for the reversal of the Ballantyne syndrome despite the fetal hydrops persistence.
巴兰坦综合征（或镜像综合征）是一种与胎儿水肿相关的妊娠蛋白尿性高血压。本报告描述了一例尽管胎儿水肿持续存在，但巴兰坦综合征仍出现逆转的病例。
一名24岁女性在妊娠28 2/7周时出现胎儿胎盘水肿。经脐静脉穿刺证实存在严重的Rh(D)同种免疫和胎儿溶血性贫血（胎儿血细胞比容15.4%），并进行了宫内输血。她还出现了高血压（160/100 mmHg）、水肿和蛋白尿（845 mg/天）。经过四次宫内输血后，血压恢复正常；尿蛋白不显著，水肿完全消失。胎儿水肿持续至妊娠32周分娩，但胎盘水肿有部分减轻。
尽管胎儿水肿持续存在，但胎盘水肿的全部或部分减轻可能是巴兰坦综合征逆转的原因。
