Wu Amy P, Davidson Terence
Division of Otolaryngology-Head and Neck Surgery, University of California-San Diego Medical Center, San Diego, California, USA.
Am J Rhinol. 2008 Nov-Dec;22(6):606-7. doi: 10.2500/ajr.2008.22.3238.
The presumed pathogenesis of posttraumatic anosmia is stretching or shearing of the olfactory nerves in a coup-contracoup head contusion. Direct injury to the brain is an alternate mechanism of injury. In this study we report a case where direct injury to the brain is the probable mechanism of injury.
A case report was performed.
A 55-year-old man presented with loss of smell beginning 1 month after a closed head injury with loss of consciousness. The MRI showed posttraumatic scarring in the region of the olfactory bulbs.
This case suggests that central nervous system injury to the olfactory bulbs and tracts may be a mechanism of posttraumatic anosmia.
创伤后嗅觉丧失的推测发病机制是在对冲性头部挫伤中嗅神经的拉伸或剪切。脑的直接损伤是另一种损伤机制。在本研究中，我们报告了一例脑的直接损伤可能是损伤机制的病例。
进行了一例病例报告。
一名55岁男性在闭合性头部损伤并伴有意识丧失1个月后出现嗅觉丧失。MRI显示嗅球区域有创伤后瘢痕形成。
该病例提示嗅球和嗅束的中枢神经系统损伤可能是创伤后嗅觉丧失的一种机制。
