Sumitomo Naokata, Fukuhara Junji, Mugishima Hideo, Sugi Kaoru
Department of Pediatrics and Child Health, Nihon University School of Medicine, Tokyo, Japan.
J Cardiovasc Electrophysiol. 2009 Jul;20(7):818-21. doi: 10.1111/j.1540-8167.2008.01404.x. Epub 2009 Jan 16.
This is a rare case of antidromic reciprocating tachycardia developing 8 years after successful catheter ablation.
A 15-year-old girl had recurrence of palpitations 8 years after the ablation of manifest right posteroseptal accessory pathway. Atrial burst pacing revealed Wenckebach atrioventricular conduction with preexcitation. Wide QRS tachycardia with identical morphology to sinus rhythm associated with retrograde His potential recorded immediately after the V-wave was induced by isoproterenol infusion. Atrial premature stimulus applied at the identical timing of His potential advanced the subsequent ventricular beat and His potential.
Catheter ablation may produce decremental accessory pathway conduction and rarely cause antidromic atrioventricular reciprocating tachycardia. This may be explained by a presence of "de novo" accessory pathway with decremental conduction properties that became manifest after the first ablation.
这是一例在成功进行导管消融术后8年发生逆向折返性心动过速的罕见病例。
一名15岁女孩在显性右后间隔旁道消融术后8年出现心悸复发。心房猝发起搏显示文氏房室传导伴预激。静脉滴注异丙肾上腺素诱发了宽QRS心动过速,其形态与窦性心律相同,且在V波后立即记录到逆行希氏束电位。在希氏束电位相同时间施加的房性期前刺激使随后的室性搏动和希氏束电位提前。
导管消融可能导致旁道传导递减,且很少引起逆向房室折返性心动过速。这可能是由于存在具有递减传导特性的“新生”旁道,在首次消融后显现出来。