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Possible mechanisms of peritoneal tissue-oxygen tension changes during CO2-pneumoperitoneum: the role of design, methodology and animal models.

作者信息

Mynbaev Ospan A, Corona Roberta

机构信息

Moscow State University of Medicine and Dentistry, Delegatskaya str. 20/1 127473, Moscow, Russian Federation.

出版信息

Hum Reprod. 2009 Jun;24(6):1242-6. doi: 10.1093/humrep/dep025. Epub 2009 Mar 3.

DOI:10.1093/humrep/dep025
PMID:19258347
Abstract
摘要

相似文献

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Possible mechanisms of peritoneal tissue-oxygen tension changes during CO2-pneumoperitoneum: the role of design, methodology and animal models.
Hum Reprod. 2009 Jun;24(6):1242-6. doi: 10.1093/humrep/dep025. Epub 2009 Mar 3.
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Peritoneal tissue-oxygen tension during a carbon dioxide pneumoperitoneum in a mouse laparoscopic model with controlled respiratory support.在具有可控呼吸支持的小鼠腹腔镜模型中，二氧化碳气腹期间的腹膜组织氧张力。
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Effect of adding more than 3% oxygen to carbon dioxide pneumoperitoneum on adhesion formation in a laparoscopic mouse model.在腹腔镜小鼠模型中，向二氧化碳气腹添加超过3%的氧气对粘连形成的影响。
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Carbon dioxide pneumoperitoneum, intraperitoneal pressure, and peritoneal tissue hypoxia: a mouse study with controlled respiratory support.二氧化碳气腹、腹腔内压和腹膜组织缺氧：一项具有控制性呼吸支持的小鼠研究。
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Comment on "Evidence for Negative Effects of Elevated Intra-Abdominal Pressure on Pulmonary Mechanics and Oxidative Stress".关于《腹内压升高对肺力学和氧化应激的负面影响的证据》的评论
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Severe inflammatory reaction induced by peritoneal trauma is the key driving mechanism of postoperative adhesion formation.
腹膜创伤引起的严重炎症反应是术后粘连形成的关键驱动机制。
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Carbon dioxide pneumoperitoneum, intraperitoneal pressure, and peritoneal tissue hypoxia: a mouse study with controlled respiratory support.二氧化碳气腹、腹腔内压和腹膜组织缺氧：一项具有控制性呼吸支持的小鼠研究。
Surg Endosc. 2010 Nov;24(11):2871-80. doi: 10.1007/s00464-010-1069-z. Epub 2010 Apr 24.