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本文引用的文献

1
Treatment of liver ischemia-reperfusion injury by limiting thrombospondin-1/CD47 signaling.通过限制血小板反应蛋白-1/CD47信号通路治疗肝脏缺血再灌注损伤
Surgery. 2008 Nov;144(5):752-61. doi: 10.1016/j.surg.2008.07.009. Epub 2008 Sep 2.
2
H2S as a physiologic vasorelaxant: hypertension in mice with deletion of cystathionine gamma-lyase.硫化氢作为一种生理性血管舒张剂：胱硫醚γ-裂解酶缺失小鼠的高血压
Science. 2008 Oct 24;322(5901):587-90. doi: 10.1126/science.1162667.
3
The inhibition of mitochondrial cytochrome oxidase by the gases carbon monoxide, nitric oxide, hydrogen cyanide and hydrogen sulfide: chemical mechanism and physiological significance.一氧化碳、一氧化氮、氰化氢和硫化氢对线粒体细胞色素氧化酶的抑制作用：化学机制与生理意义
J Bioenerg Biomembr. 2008 Oct;40(5):533-9. doi: 10.1007/s10863-008-9166-6. Epub 2008 Oct 7.
4
Nitroxyl (HNO): the Cinderella of the nitric oxide story.硝酰基（HNO）：一氧化氮故事中的灰姑娘。
Trends Pharmacol Sci. 2008 Dec;29(12):601-8. doi: 10.1016/j.tips.2008.08.005. Epub 2008 Oct 1.
5
Tumor-vascular interactions and tumor dormancy.肿瘤-血管相互作用与肿瘤休眠
APMIS. 2008 Jul-Aug;116(7-8):569-85. doi: 10.1111/j.1600-0463.2008.01213.x.
6
The -786T>C promoter polymorphism of the NOS3 gene is associated with prostate cancer progression.一氧化氮合酶3基因（NOS3）-786T>C启动子多态性与前列腺癌进展相关。
BMC Cancer. 2008 Sep 29;8:273. doi: 10.1186/1471-2407-8-273.
7
Thrombospondin-1 and CD47 limit cell and tissue survival of radiation injury.血小板反应蛋白-1和CD47限制辐射损伤的细胞和组织存活。
Am J Pathol. 2008 Oct;173(4):1100-12. doi: 10.2353/ajpath.2008.080237. Epub 2008 Sep 11.
8
Angiopoietin-2 stimulates blood flow recovery after femoral artery occlusion by inducing inflammation and arteriogenesis.血管生成素-2通过诱导炎症反应和动脉生成来刺激股动脉闭塞后血流的恢复。
Arterioscler Thromb Vasc Biol. 2008 Nov;28(11):1989-95. doi: 10.1161/ATVBAHA.108.175463. Epub 2008 Sep 4.
9
Thrombospondin 1 and vasoactive agents indirectly alter tumor blood flow.血小板反应蛋白1和血管活性药物间接改变肿瘤血流。
Neoplasia. 2008 Aug;10(8):886-96. doi: 10.1593/neo.08264.
10
Modes of resistance to anti-angiogenic therapy.抗血管生成疗法的耐药模式。
Nat Rev Cancer. 2008 Aug;8(8):592-603. doi: 10.1038/nrc2442.

Molecular regulation of tumor angiogenesis and perfusion via redox signaling.

作者信息

Miller Thomas W, Isenberg Jeff S, Roberts David D

机构信息

Laboratory of Pathology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA.

出版信息

Chem Rev. 2009 Jul;109(7):3099-124. doi: 10.1021/cr8005125.

DOI:10.1021/cr8005125
PMID:19374334
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2801866/
Abstract
摘要