Lieberman Philip
Cognitive and Linguistic Sciences, Brown University, Providence, RI 02912, USA.
Cell. 2009 May 29;137(5):800-2. doi: 10.1016/j.cell.2009.05.013.
Using a mouse model, Enard et al. (2009) show that the human form of the FOXP2 gene increases synaptic plasticity and dendrite connectivity in the basal ganglia. These results partly explain the enhanced capability of cortico-basal ganglia circuits in the human brain that regulate critical aspects of language, cognition, and motor control.
埃纳尔等人(2009年)利用小鼠模型表明,人类形式的FOXP2基因可增强基底神经节中的突触可塑性和树突连接性。这些结果部分解释了人类大脑中皮质-基底神经节回路在调节语言、认知和运动控制等关键方面能力的增强。