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4:2传导模式的房室传导阻滞:其机制是什么?

Atrioventricular block with 4:2 conduction pattern: what is the mechanism?

作者信息

Jastrzebski Marek, Kukla Piotr

机构信息

1st Department of Cardiology and Hypertension, University Hospital, ul. Kopernika 17, Krakow, Poland.

出版信息

J Electrocardiol. 2009 Nov-Dec;42(6):684-6. doi: 10.1016/j.jelectrocard.2009.06.001. Epub 2009 Jun 30.

DOI:10.1016/j.jelectrocard.2009.06.001
PMID:19570547
Abstract

We present the case of a 72-year-old man who was admitted because of acute ST-segment elevation myocardial infarction and presented with second-degree atrioventricular block with 4:2 conduction. We propose 3 alternative explanations for such an unusual conduction pattern: (1) supernormal conduction, (2) 2-level block with 4:1 conduction ratio in the upper level and 3:2 conduction ratio in the lower level, and (3) presence of 2 populations of Purkinje cells in the remaining, critically injured fascicle, one with the phase-3 block and the other with slow diastolic depolarization, leading, during a pause after the first nonconducted P, to phase-4 block.

摘要

我们报告了一例72岁男性患者,因急性ST段抬高型心肌梗死入院,伴有二度房室传导阻滞,传导比例为4:2。对于这种不寻常的传导模式,我们提出了3种可能的解释:(1)超常传导;(2)二级阻滞,上级传导比例为4:1,下级传导比例为3:2;(3)在剩余严重受损的束支中存在2组浦肯野细胞,一组具有3相阻滞,另一组具有缓慢舒张期去极化,在第一个未下传的P波后的间歇期导致4相阻滞。

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Atrioventricular block with 4:2 conduction pattern: what is the mechanism?4:2传导模式的房室传导阻滞:其机制是什么?
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引用本文的文献

1
An analysis of a 4:2 atrioventricular block, a rare occurrence.对4:2房室传导阻滞的分析,这是一种罕见情况。
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