前列腺素E对鼠肝炎病毒3型感染的保护作用机制：对巨噬细胞产生肿瘤坏死因子、促凝血活性和白三烯B4的影响 - Suppr | 超能文献

Mechanism of protective effect of prostaglandin E in murine hepatitis virus strain 3 infection: effects on macrophage production of tumour necrosis factor, procoagulant activity and leukotriene B4.

作者信息

Sinclair S, Abecassis M, Wong P Y, Romaschin A, Fung L S, Levy G

机构信息

Department of Medicine, University of Toronto, Canada.

出版信息

Adv Exp Med Biol. 1990;276:533-42. doi: 10.1007/978-1-4684-5823-7_74.

DOI:10.1007/978-1-4684-5823-7_74

Abstract

摘要

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Mechanism of protective effect of prostaglandin E in murine hepatitis virus strain 3 infection: effects on macrophage production of tumour necrosis factor, procoagulant activity and leukotriene B4.前列腺素E对鼠肝炎病毒3型感染的保护作用机制：对巨噬细胞产生肿瘤坏死因子、促凝血活性和白三烯B4的影响

Adv Exp Med Biol. 1990;276:533-42. doi: 10.1007/978-1-4684-5823-7_74.

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Pattern of disease after murine hepatitis virus strain 3 infection correlates with macrophage activation and not viral replication.鼠肝炎病毒3型感染后的疾病模式与巨噬细胞激活相关，而非病毒复制。

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16, 16 Dimethyl prostaglandin E2 prevents the development of fulminant hepatitis and blocks the induction of monocyte/macrophage procoagulant activity after murine hepatitis virus strain 3 infection.16,16-二甲基前列腺素E2可预防暴发性肝炎的发生，并在鼠肝炎病毒3型感染后阻断单核细胞/巨噬细胞促凝活性的诱导。

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Effect of eicosanoids on induction of procoagulant activity by murine hepatitis virus strain 3 in vitro.类花生酸对小鼠肝炎病毒3型体外诱导促凝活性的影响。

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Prostaglandin E2 (PGE2) alters the pathogenesis of MHV-3 infection in susceptible BALB/cJ mice.前列腺素E2（PGE2）可改变易感BALB/cJ小鼠中MHV - 3感染的发病机制。

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Prostaglandin E2 prevents fulminant hepatitis and the induction of procoagulant activity in susceptible animals.前列腺素E2可预防易感动物发生暴发性肝炎并抑制促凝血活性的诱导。

Transplant Proc. 1987 Feb;19(1 Pt 2):1103-5.

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Role of macrophage procoagulant activity in mouse hepatitis virus (MHV) infection: studies using T cell MHV-3 clones and monoclonal antibody 3D4.3.巨噬细胞促凝活性在小鼠肝炎病毒（MHV）感染中的作用：使用T细胞MHV - 3克隆和单克隆抗体3D4.3的研究

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Lymphocyte-instructed monocyte induction of the coagulation pathways parallels the induction of hepatitis by the murine hepatitis virus.淋巴细胞指导的单核细胞对凝血途径的诱导与鼠肝炎病毒诱导的肝炎过程相似。

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Acquired immunity of A/J mice to mouse hepatitis virus 3 infection: dependence on interferon-gamma synthesis and macrophage sensitivity to interferon-gamma.A/J小鼠对小鼠肝炎病毒3型感染的获得性免疫：依赖于γ干扰素的合成以及巨噬细胞对γ干扰素的敏感性。

J Gen Virol. 1991 Jun;72 ( Pt 6):1317-22. doi: 10.1099/0022-1317-72-6-1317.

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Resistance of naive mice to murine hepatitis virus strain 3 requires development of a Th1, but not a Th2, response, whereas pre-existing antibody partially protects against primary infection.未接触过抗原的小鼠对鼠肝炎病毒3型的抵抗力需要Th1应答的产生，而非Th2应答，而预先存在的抗体可部分保护小鼠免受初次感染。

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Enhanced tumor necrosis factor alpha in coronavirus but not in paracetamol-induced acute hepatic necrosis in mice.冠状病毒感染所致而非对乙酰氨基酚诱导的小鼠急性肝坏死中肿瘤坏死因子α水平升高。

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Monoclonal antiprothrombinase (3D4.3) prevents mortality from murine hepatitis virus (MHV-3) infection.单克隆抗凝血酶原酶（3D4.3）可预防小鼠感染鼠肝炎病毒（MHV-3）后的死亡。

J Exp Med. 1992 Sep 1;176(3):689-97. doi: 10.1084/jem.176.3.689.