前列腺素E2（PGE2）可改变易感BALB/cJ小鼠中MHV - 3感染的发病机制。 - Suppr | 超能文献

Prostaglandin E2 (PGE2) alters the pathogenesis of MHV-3 infection in susceptible BALB/cJ mice.

作者信息

Abecassis M, Falk J, Dindzans V, Lopatin W, Makowka L, Levy G, Falk R

机构信息

Toronto General Hospital, Ontario, Canada.

出版信息

Adv Exp Med Biol. 1987;218:465-6. doi: 10.1007/978-1-4684-1280-2_58.

DOI:10.1007/978-1-4684-1280-2_58

Abstract

摘要

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Prostaglandin E2 (PGE2) alters the pathogenesis of MHV-3 infection in susceptible BALB/cJ mice.前列腺素E2（PGE2）可改变易感BALB/cJ小鼠中MHV - 3感染的发病机制。

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16, 16 Dimethyl prostaglandin E2 prevents the development of fulminant hepatitis and blocks the induction of monocyte/macrophage procoagulant activity after murine hepatitis virus strain 3 infection.16,16-二甲基前列腺素E2可预防暴发性肝炎的发生，并在鼠肝炎病毒3型感染后阻断单核细胞/巨噬细胞促凝活性的诱导。

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A 2',5'-oligoadenylate analogue inhibits murine hepatitis virus strain 3 (MHV-3) replication in vitro but does not reduce MHV-3-related mortality or induction of procoagulant activity in susceptible mice.一种2',5'-寡腺苷酸类似物在体外可抑制鼠肝炎病毒3型（MHV-3）的复制，但不能降低易感小鼠中与MHV-3相关的死亡率或促凝血活性的诱导。

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