The effect of Li on the glucagon-sensitive adenylate cyclase in vivo in man.
作者信息
Ebstein R P, Kara T, Belmaker R H
出版信息
Acta Pharmacol Toxicol (Copenh). 1977 Jul;41(1):80-3. doi: 10.1111/j.1600-0773.1977.tb02126.x.
Abstract
摘要
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The effect of Li on the glucagon-sensitive adenylate cyclase in vivo in man.锂对人体体内胰高血糖素敏感性腺苷酸环化酶的作用。
Acta Pharmacol Toxicol (Copenh). 1977 Jul;41(1):80-3. doi: 10.1111/j.1600-0773.1977.tb02126.x.
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The hepatic glucose-mobilizing effect of glucagon is not mediated by cyclic AMP most of the time.大多数情况下，胰高血糖素的肝脏葡萄糖动员作用并非由环磷酸腺苷介导。
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The rapid desensitization of glucagon-stimulated adenylate cyclase is a cyclic AMP-independent process that can be mimicked by hormones which stimulate inositol phospholipid metabolism.胰高血糖素刺激的腺苷酸环化酶的快速脱敏是一个不依赖环磷酸腺苷的过程，可被刺激肌醇磷脂代谢的激素模拟。
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Elevation of intracellular cyclic AMP and stimulation of adenylate cyclase activity by vasoactive intestinal peptide and glucagon in the retinal pigment epithelium.
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Glucagon stimulates adenylate cyclase through GR2 glucagon receptors: a process which can be attenuated by glucagon stimulating inositol phospholipid metabolism through GR1 glucagon receptors.
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Lithium inhibition of the adenosine-induced increase of adenylate cyclase activity.锂对腺苷诱导的腺苷酸环化酶活性增加的抑制作用。
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