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Ischaemic preconditioning and postconditioning do not affect adenosine A(1) and A (2A) receptor sensitivity.

作者信息

Riksen Niels P, Wynne Abigail, Yellon Derek M, Hausenloy Derek J

出版信息

Cardiovasc Drugs Ther. 2009 Oct;23(5):415-7. doi: 10.1007/s10557-009-6196-1.

DOI:10.1007/s10557-009-6196-1
PMID:19784766
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2788144/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b694/2788144/d1a765b1924e/10557_2009_6196_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b694/2788144/b0e3f830dc05/10557_2009_6196_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b694/2788144/d1a765b1924e/10557_2009_6196_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b694/2788144/b0e3f830dc05/10557_2009_6196_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b694/2788144/d1a765b1924e/10557_2009_6196_Fig2_HTML.jpg

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本文引用的文献

1
Targeted deletion of A2A adenosine receptors attenuates the protective effects of myocardial postconditioning.A2A 腺苷受体的靶向缺失减弱了心肌后适应的保护作用。
Am J Physiol Heart Circ Physiol. 2007 Oct;293(4):H2523-9. doi: 10.1152/ajpheart.00612.2007. Epub 2007 Aug 3.
2
Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b-dependent signaling during early reperfusion.蛋白激酶C通过在早期再灌注期间增加腺苷A2b依赖性信号传导的敏感性来保护预处理的兔心脏。
J Mol Cell Cardiol. 2007 Sep;43(3):262-71. doi: 10.1016/j.yjmcc.2007.05.016. Epub 2007 May 24.
3
Endogenous adenosine protects preconditioned heart during early minutes of reperfusion by activating Akt.
内源性腺苷通过激活Akt在再灌注早期保护经预处理的心脏。
Am J Physiol Heart Circ Physiol. 2006 Jan;290(1):H441-9. doi: 10.1152/ajpheart.00589.2005. Epub 2005 Sep 9.
4
Postconditioning reduces infarct size via adenosine receptor activation by endogenous adenosine.缺血后适应通过内源性腺苷激活腺苷受体来减小梗死面积。
Cardiovasc Res. 2005 Jul 1;67(1):124-33. doi: 10.1016/j.cardiores.2005.02.015. Epub 2005 Apr 1.
5
Blockade of nucleoside transport is required for delivery of intraarterial adenosine into the interstitium: relevance to therapeutic preconditioning in humans.动脉内注射腺苷进入间质需要核苷转运的阻断:与人类治疗性预处理的相关性
Circulation. 2003 Nov 25;108(21):2631-5. doi: 10.1161/01.CIR.0000101927.70100.41. Epub 2003 Nov 17.
6
Comparative study of AMP579 and adenosine in inhibition of neutrophil-mediated vascular and myocardial injury during 24 h of reperfusion.AMP579与腺苷在再灌注24小时期间对中性粒细胞介导的血管和心肌损伤抑制作用的比较研究。
Cardiovasc Res. 2000 Aug;47(2):294-305. doi: 10.1016/s0008-6363(00)00115-2.
7
Effect of adenosine therapy at reperfusion on myocardial infarct size in dogs.再灌注时腺苷治疗对犬心肌梗死面积的影响。
Cardiovasc Res. 1996 May;31(5):711-8. doi: 10.1016/0008-6363(95)00235-9.
8
Adenosine infusion during early reperfusion failed to limit myocardial infarct size in a collateral deficient species.在早期再灌注期间输注腺苷未能在侧支循环不足的物种中限制心肌梗死面积。
Cardiovasc Res. 1991 Nov;25(11):943-9. doi: 10.1093/cvr/25.11.943.