Faculty of Management, Tel Aviv University, Tel Aviv, Israel.
J Occup Health Psychol. 2009 Oct;14(4):349-64. doi: 10.1037/a0015283.
The authors investigated the direct and interactive effects of the job demand- control-support (JDC-S) model's components on subsequent changes in high-density lipoprotein cholesterol, low-density lipoprotein cholesterol, and triglycerides (TRI) separately for male and female employees. In contrast to all 14 past studies on these relationships, the authors used a longitudinal design. Study participants (N = 1,137, 66% men) were all apparently healthy employees who underwent a routine health check at 2 points in time (Time 1 and Time 2) about 22 months apart. In these analyses, the authors controlled for the Time 1 level of each criterion and for other confounders. Most of the direct and moderating effects found did not support the predictions of the JDC-S model; this finding is in agreement with the majority of past cross-sectional studies. The authors did not find any evidence supporting the existence of a reverse causation for either of the components of the JDC-S model. The authors suggest that serum lipids may not be a physiological mechanism mediating the effects of the JDC-S model on atherosclerotic diseases.
作者分别研究了工作需求-控制-支持（JDC-S）模型的组成部分对男性和女性员工随后高密度脂蛋白胆固醇、低密度脂蛋白胆固醇和甘油三酯（TRI）变化的直接和交互影响。与过去关于这些关系的所有 14 项研究不同，作者采用了纵向设计。研究参与者（N=1137，66%为男性）均为健康状况良好的员工，他们在大约 22 个月的两次时间（时间 1 和时间 2）进行了常规健康检查。在这些分析中，作者控制了每个标准的时间 1 水平和其他混杂因素。大多数发现的直接和调节作用并不支持 JDC-S 模型的预测；这一发现与过去大多数横断面研究一致。作者没有发现任何证据支持 JDC-S 模型的组成部分存在反向因果关系。作者认为，血清脂质可能不是介导 JDC-S 模型对动脉粥样硬化疾病影响的生理机制。
