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神经毒性胺1-甲基-4-苯基吡啶鎓(MPP+)和呼吸链抑制剂粉蝶霉素A的抑制位点相同的证据。

Evidence that the inhibition sites of the neurotoxic amine 1-methyl-4-phenylpyridinium (MPP+) and of the respiratory chain inhibitor piericidin A are the same.

作者信息

Ramsay R R, Krueger M J, Youngster S K, Singer T P

机构信息

Department of Biochemistry and Biophysics, University of California, San Francisco 94143.

出版信息

Biochem J. 1991 Jan 15;273(Pt 2)(Pt 2):481-4. doi: 10.1042/bj2730481.

Abstract

1-Methyl-4-phenylpyridinium (MPP+), the neurotoxic bioactivation product of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), interrupts mitochondrial electron transfer at the NADH dehydrogenase-ubiquinone junction, as do the respiratory chain inhibitors rotenone, piericidin A and barbiturates. Proof that these classical respiratory chain inhibitors and MPP+ react at the same site in the complex NADH dehydrogenase molecule has been difficult to obtain because none of these compounds bind covalently to the target. The 4'-alkyl derivatives of MPP+ inhibit NADH oxidation in submitochondrial particles at much lower concentrations than does MPP+ itself, but still dissociate on washing the membrane preparations, with consequent re-activation of the enzyme. The MPP+ analogues with short alkyl chains prevent the binding of 14C-labelled piericidin A to the membrane and thus must act at the same site, but analogues with alkyl chains longer than heptyl do not prevent binding of [14C]piericidin.

摘要

1-甲基-4-苯基吡啶鎓(MPP⁺)是1-甲基-4-苯基-1,2,3,6-四氢吡啶(MPTP)的神经毒性生物活化产物,它在NADH脱氢酶-泛醌连接处干扰线粒体电子传递,呼吸链抑制剂鱼藤酮、杀粉蝶菌素A和巴比妥类药物也有同样的作用。由于这些化合物均不能与靶标共价结合,因此很难证明这些经典的呼吸链抑制剂和MPP⁺在复合NADH脱氢酶分子的同一部位发生反应。MPP⁺的4'-烷基衍生物在比MPP⁺本身低得多的浓度下就能抑制亚线粒体颗粒中的NADH氧化,但在洗涤膜制剂时仍会解离,从而使酶重新激活。具有短烷基链的MPP⁺类似物可阻止¹⁴C标记的杀粉蝶菌素A与膜结合,因此必定作用于同一部位,但烷基链长于庚基的类似物则不能阻止[¹⁴C]杀粉蝶菌素的结合。

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