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A CASE OF MULTIPLE MYELOMA.

作者信息

Wright James H

出版信息

J Boston Soc Med Sci. 1900 Apr 10;4(8):195-204.5.

PMID:19971313
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2121902/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/ca8243470269/jbsms00014-0026-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/dbbb2015fe64/jbsms00014-0025-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/d516cfb54b65/jbsms00014-0025-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/c8011dc491d2/jbsms00014-0024-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/019a042bd276/jbsms00014-0024-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/1799a6b6655d/jbsms00014-0024-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/208cffd782ec/jbsms00014-0026-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/fa35dc98e7a3/jbsms00014-0026-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/ca8243470269/jbsms00014-0026-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/dbbb2015fe64/jbsms00014-0025-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/d516cfb54b65/jbsms00014-0025-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/c8011dc491d2/jbsms00014-0024-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/019a042bd276/jbsms00014-0024-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/1799a6b6655d/jbsms00014-0024-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/208cffd782ec/jbsms00014-0026-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/fa35dc98e7a3/jbsms00014-0026-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/bf70/2121902/ca8243470269/jbsms00014-0026-c.jpg

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A CASE OF MULTIPLE MYELOMA.一例多发性骨髓瘤病例。
J Boston Soc Med Sci. 1900 Apr 10;4(8):195-204.5.
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[IgD (lambda) multiple myeloma associated with myelofibrosis: an isolated case of nuclear physicist].[与骨髓纤维化相关的IgD（λ）多发性骨髓瘤：一名核物理学家的孤立病例]
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引用本文的文献

1
Inhibition of dual-specificity tyrosine phosphorylation-regulated kinase 2 perturbs 26S proteasome-addicted neoplastic progression.双重特异性酪氨酸磷酸化调节激酶 2 的抑制扰乱了依赖 26S 蛋白酶体的肿瘤进展。
Proc Natl Acad Sci U S A. 2019 Dec 3;116(49):24881-24891. doi: 10.1073/pnas.1912033116. Epub 2019 Nov 21.
2
Cell of Origin and Genetic Alterations in the Pathogenesis of Multiple Myeloma.多发性骨髓瘤发病机制中的起源细胞和遗传改变。
Front Immunol. 2019 May 21;10:1121. doi: 10.3389/fimmu.2019.01121. eCollection 2019.
3
Signaling Interplay between Bone Marrow Adipose Tissue and Multiple Myeloma cells.
骨髓脂肪组织与多发性骨髓瘤细胞之间的信号相互作用。
Front Endocrinol (Lausanne). 2016 Jun 17;7:67. doi: 10.3389/fendo.2016.00067. eCollection 2016.
4
The flowering of pathology as a medical discipline in Boston, 1892-c.1950: W.T. Councilman, FB Mallory, JH Wright, SB Wolbach and their descendants.病理学作为一门医学学科在波士顿的蓬勃发展，1892年至约1950年：W.T. 康西尔曼、F.B. 马洛里、J.H. 赖特、S.B. 沃尔巴赫及其后人。
Mod Pathol. 2016 Sep;29(9):944-61. doi: 10.1038/modpathol.2016.91. Epub 2016 Jun 17.
5
"Punched out" multiple myeloma lytic lesions in the skull.颅骨中出现的多发性骨髓瘤“凿孔样”溶骨性病变。
Autops Case Rep. 2016 Mar 30;6(1):7-9. doi: 10.4322/acr.2016.020. eCollection 2016 Jan-Mar.
6
Multiple myelomatosis; a clinico-pathological review, with a report of a case of myeloblastic type.多发性骨髓瘤；临床病理综述，并报告一例成髓细胞型病例。
Postgrad Med J. 1948 May;24(271):269-82. doi: 10.1136/pgmj.24.271.269.