相似文献
1
The dangers of transcription.
Cell. 2009 Dec 11;139(6):1047-9. doi: 10.1016/j.cell.2009.11.037.
2
Histone modifications predispose genome regions to breakage and translocation.
Genes Dev. 2015 Jul 1;29(13):1393-402. doi: 10.1101/gad.262170.115. Epub 2015 Jun 23.
3
Nuclear receptor-induced chromosomal proximity and DNA breaks underlie specific translocations in cancer.
Cell. 2009 Dec 11;139(6):1069-83. doi: 10.1016/j.cell.2009.11.030.
4
Dynamics of double strand breaks and chromosomal translocations.
Mol Cancer. 2014 Nov 18;13:249. doi: 10.1186/1476-4598-13-249.
5
Assembly of methylated KDM1A and CHD1 drives androgen receptor-dependent transcription and translocation.
Nat Struct Mol Biol. 2016 Feb;23(2):132-9. doi: 10.1038/nsmb.3153. Epub 2016 Jan 11.
6
Dynamics of Double-Strand Breaks: Implications for the Formation of Chromosome Translocations.
Adv Exp Med Biol. 2018;1044:27-38. doi: 10.1007/978-981-13-0593-1_3.
7
TDP2 suppresses chromosomal translocations induced by DNA topoisomerase II during gene transcription.
Nat Commun. 2017 Aug 10;8(1):233. doi: 10.1038/s41467-017-00307-y.
8
Snaps and mends: DNA breaks and chromosomal translocations.
FEBS J. 2015 Jul;282(14):2627-45. doi: 10.1111/febs.13311. Epub 2015 May 19.
9
Anchoring Chromatin Loops to Cancer.
Dev Cell. 2017 Aug 7;42(3):209-211. doi: 10.1016/j.devcel.2017.07.013.
10
Antigen receptor diversification and chromosome translocations.
Nat Immunol. 2007 Aug;8(8):801-8. doi: 10.1038/ni1498.
引用本文的文献
1
Recent advances on the progressive mechanism and therapy in castration-resistant prostate cancer.
Onco Targets Ther. 2018 May 28;11:3167-3178. doi: 10.2147/OTT.S159777. eCollection 2018.
2
Translational and clinical implications of the genetic landscape of prostate cancer.
Nat Rev Clin Oncol. 2016 Oct;13(10):597-610. doi: 10.1038/nrclinonc.2016.76. Epub 2016 Jun 1.
3
Targeting the adaptive molecular landscape of castration-resistant prostate cancer.
EMBO Mol Med. 2015 Jul;7(7):878-94. doi: 10.15252/emmm.201303701.
4
The biogenesis of chromosome translocations.
Nat Cell Biol. 2014 Apr;16(4):293-300. doi: 10.1038/ncb2941.
5
The cellular etiology of chromosome translocations.
Curr Opin Cell Biol. 2013 Jun;25(3):357-64. doi: 10.1016/j.ceb.2013.02.015. Epub 2013 Mar 14.
6
Deubiquitination of NF-κB by Ubiquitin-Specific Protease-7 promotes transcription.
Proc Natl Acad Sci U S A. 2013 Jan 8;110(2):618-23. doi: 10.1073/pnas.1208446110. Epub 2012 Dec 24.
7
Correct end use during end joining of multiple chromosomal double strand breaks is influenced by repair protein RAD50, DNA-dependent protein kinase DNA-PKcs, and transcription context.
J Biol Chem. 2011 Dec 9;286(49):42470-42482. doi: 10.1074/jbc.M111.309252. Epub 2011 Oct 24.
8
Androgen receptor-driven chromatin looping in prostate cancer.
Trends Endocrinol Metab. 2011 Dec;22(12):474-80. doi: 10.1016/j.tem.2011.07.006. Epub 2011 Aug 31.
本文引用的文献
1
Nuclear receptor-induced chromosomal proximity and DNA breaks underlie specific translocations in cancer.
Cell. 2009 Dec 11;139(6):1069-83. doi: 10.1016/j.cell.2009.11.030.
2
Induced chromosomal proximity and gene fusions in prostate cancer.
Science. 2009 Nov 27;326(5957):1230. doi: 10.1126/science.1178124. Epub 2009 Oct 29.
3
Gene deregulation and spatial genome reorganization near breakpoints prior to formation of translocations in anaplastic large cell lymphoma.
Proc Natl Acad Sci U S A. 2009 Apr 7;106(14):5831-6. doi: 10.1073/pnas.0900912106. Epub 2009 Mar 25.
4
The emerging role of nuclear architecture in DNA repair and genome maintenance.
Nat Rev Mol Cell Biol. 2009 Apr;10(4):243-54. doi: 10.1038/nrm2651. Epub 2009 Mar 11.
5
AID is required for the chromosomal breaks in c-myc that lead to c-myc/IgH translocations.
Cell. 2008 Dec 12;135(6):1028-38. doi: 10.1016/j.cell.2008.09.062.
6
Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer.
Science. 2005 Oct 28;310(5748):644-8. doi: 10.1126/science.1117679.
Zotero 插件