相似文献
1
Mitotic chromosomal instability and cancer: mouse modelling of the human disease.有丝分裂染色体不稳定性与癌症：人类疾病的小鼠模型。
Nat Rev Cancer. 2010 Feb;10(2):102-15. doi: 10.1038/nrc2781.
2
Boveri revisited: chromosomal instability, aneuploidy and tumorigenesis.重访博韦里：染色体不稳定、非整倍体与肿瘤发生
Nat Rev Mol Cell Biol. 2009 Jul;10(7):478-87. doi: 10.1038/nrm2718.
3
Mechanisms of chromosomal instability.染色体不稳定性的机制。
Curr Biol. 2010 Mar 23;20(6):R285-95. doi: 10.1016/j.cub.2010.01.034.
4
Deregulation of the centrosome cycle and the origin of chromosomal instability in cancer.中心体周期失调与癌症中染色体不稳定性的起源
Adv Exp Med Biol. 2005;570:393-421. doi: 10.1007/1-4020-3764-3_14.
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The role of chromosome missegregation in cancer development: a theoretical approach using agent-based modelling.染色体错误分离在癌症发展中的作用：基于代理的建模理论方法。
PLoS One. 2013 Aug 26;8(8):e72206. doi: 10.1371/journal.pone.0072206. eCollection 2013.
6
Examining the link between chromosomal instability and aneuploidy in human cells.研究人类细胞中染色体不稳定性与非整倍体之间的联系。
J Cell Biol. 2008 Feb 25;180(4):665-72. doi: 10.1083/jcb.200712029. Epub 2008 Feb 18.
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Simultaneous Aurora-A/STK15 overexpression and centrosome amplification induce chromosomal instability in tumour cells with a MIN phenotype.极光激酶A/丝氨酸苏氨酸激酶15（Aurora-A/STK15）同时过表达和中心体扩增会在具有微卫星不稳定性（MIN）表型的肿瘤细胞中诱导染色体不稳定。
BMC Cancer. 2007 Nov 13;7:212. doi: 10.1186/1471-2407-7-212.
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Living in CIN: Mitotic Infidelity and Its Consequences for Tumor Promotion and Suppression.生活在染色体不稳定状态：有丝分裂错误及其对肿瘤促进和抑制的影响。
Dev Cell. 2016 Dec 19;39(6):638-652. doi: 10.1016/j.devcel.2016.10.023.
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CIN By WNT: growth pathways, mitotic control and chromosomal instability in cancer.通过WNT信号通路的宫颈癌：癌症中的生长途径、有丝分裂控制和染色体不稳定性
Cell Cycle. 2006 Sep;5(18):2077-81. doi: 10.4161/cc.5.18.3282. Epub 2006 Sep 15.
10
The wages of CIN.对比剂肾病的代价
J Cell Biol. 2008 Feb 25;180(4):661-3. doi: 10.1083/jcb.200801030. Epub 2008 Feb 18.
引用本文的文献
1
Identification of targetable vulnerabilities of PLK1-overexpressing cancers by synthetic dosage lethality.通过合成剂量致死性鉴定PLK1过表达癌症的可靶向弱点。
Cell Genom. 2025 Jun 11;5(6):100876. doi: 10.1016/j.xgen.2025.100876. Epub 2025 May 9.
2
Replication stress promotes cellular transformation in Drosophila epithelium.复制应激促进果蝇上皮细胞的转化。
Cell Death Discov. 2025 Mar 12;11(1):96. doi: 10.1038/s41420-025-02383-2.
3
Chromosome Missegregation Triggers Tumor Cell Pyroptosis and Enhances Anti-Tumor Immunotherapy in Colorectal Cancer.染色体错分离引发肿瘤细胞焦亡并增强结直肠癌的抗肿瘤免疫治疗
Adv Sci (Weinh). 2025 Mar;12(12):e2409769. doi: 10.1002/advs.202409769. Epub 2025 Feb 4.
4
Fine-tuning probes for fluorescence polarization binding assays of bivalent ligands against polo-like kinase 1 using full-length protein.使用全长蛋白对针对polo样激酶1的二价配体进行荧光偏振结合测定的微调探针。
Bioorg Med Chem. 2025 Mar 1;119:118055. doi: 10.1016/j.bmc.2024.118055. Epub 2024 Dec 28.
5
Role of TRIP13 in human cancer development.TRIP13 在人类癌症发展中的作用。
Mol Biol Rep. 2024 Oct 22;51(1):1088. doi: 10.1007/s11033-024-10012-x.
6
Identification of autoantibodies as potential non-invasive biomarkers for intrahepatic cholangiocarcinoma.鉴定自身抗体作为肝内胆管癌潜在的非侵入性生物标志物。
Sci Rep. 2024 Aug 28;14(1):20012. doi: 10.1038/s41598-024-70595-0.
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Mosaic variegated aneuploidy in development, ageing and cancer.发育、衰老和癌症中的镶嵌性非整倍体
Nat Rev Genet. 2024 Dec;25(12):864-878. doi: 10.1038/s41576-024-00762-6. Epub 2024 Aug 21.
8
Affinity enhancement of polo-like kinase 1 polo box domain-binding ligands by a bivalent approach using a covalent kinase-binding component.使用共价激酶结合成分通过二价方法增强polo样激酶1的polo盒结构域结合配体的亲和力。
RSC Chem Biol. 2024 Jun 4;5(8):721-728. doi: 10.1039/d4cb00031e. eCollection 2024 Jul 31.
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Application of a Fluorescence Recovery-Based Polo-Like Kinase 1 Binding Assay to Polo-Like Kinase 2 and Polo-Like Kinase 3.基于荧光恢复的 Polo 样激酶 1 结合分析法在 Polo 样激酶 2 和 Polo 样激酶 3 中的应用。
Biol Pharm Bull. 2024;47(7):1282-1287. doi: 10.1248/bpb.b24-00189.
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YY2/BUB3 Axis promotes SAC Hyperactivation and Inhibits Colorectal Cancer Progression via Regulating Chromosomal Instability.YY2/BUB3 轴通过调节染色体不稳定性促进 SAC 过度激活并抑制结直肠癌进展。
Adv Sci (Weinh). 2024 Jul;11(26):e2308690. doi: 10.1002/advs.202308690. Epub 2024 Apr 29.
本文引用的文献
1
Mad2-induced chromosome instability leads to lung tumour relapse after oncogene withdrawal.Mad2 诱导的染色体不稳定性导致致癌基因失活后肺肿瘤复发。
Nature. 2010 Mar 18;464(7287):436-40. doi: 10.1038/nature08803. Epub 2010 Feb 21.
2
Evidence that mitotic exit is a better cancer therapeutic target than spindle assembly.有证据表明，与纺锤体组装相比，有丝分裂退出是更好的癌症治疗靶点。
Cancer Cell. 2009 Oct 6;16(4):347-58. doi: 10.1016/j.ccr.2009.08.020.
3
On the karyotypic origin and evolution of cancer cells.论癌细胞的核型起源与进化。
Cancer Genet Cytogenet. 2009 Oct 15;194(2):96-110. doi: 10.1016/j.cancergencyto.2009.06.008.
4
Multipolar spindle pole coalescence is a major source of kinetochore mis-attachment and chromosome mis-segregation in cancer cells.多极纺锤体极融合是癌细胞中动粒错误附着和染色体错误分离的主要来源。
PLoS One. 2009 Aug 10;4(8):e6564. doi: 10.1371/journal.pone.0006564.
5
Comparative analysis of protein expression in differentiated thyroid tumours: a multicentre study.
J Int Med Res. 2009 May-Jun;37(3):927-38. doi: 10.1177/147323000903700339.
6
Boveri revisited: chromosomal instability, aneuploidy and tumorigenesis.重访博韦里：染色体不稳定、非整倍体与肿瘤发生
Nat Rev Mol Cell Biol. 2009 Jul;10(7):478-87. doi: 10.1038/nrm2718.
7
Loss of spindle assembly checkpoint-mediated inhibition of Cdc20 promotes tumorigenesis in mice.纺锤体组装检查点介导的Cdc20抑制作用丧失促进小鼠肿瘤发生。
J Cell Biol. 2009 Jun 15;185(6):983-94. doi: 10.1083/jcb.200904020.
8
A mechanism linking extra centrosomes to chromosomal instability.一种将额外中心体与染色体不稳定性相联系的机制。
Nature. 2009 Jul 9;460(7252):278-82. doi: 10.1038/nature08136. Epub 2009 Jun 7.
9
Integrative analysis of cyclin protein levels identifies cyclin b1 as a classifier and predictor of outcomes in breast cancer.细胞周期蛋白水平的综合分析确定细胞周期蛋白B1为乳腺癌预后的分类指标和预测指标。
Clin Cancer Res. 2009 Jun 1;15(11):3654-62. doi: 10.1158/1078-0432.CCR-08-3293. Epub 2009 May 26.
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MAD2 as a key component of mitotic checkpoint: A probable prognostic factor for gastric cancer.MAD2作为有丝分裂检查点的关键组成部分：一种可能的胃癌预后因素。
Am J Clin Pathol. 2009 Jun;131(6):793-801. doi: 10.1309/AJCPBMHHD0HFCY8W.
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