Pfizenmaier K, Starzinski-Powitz A, Wagner H, Röllinghoff M
Z Immunitatsforsch Immunobiol. 1977 Sep;153(3):268-73.
Non-infectious sendai virus renders H-2 matched target cells susceptible to the lytic effect of sendai virus immune cytotoxic T lymphocytes. This observation suggests that exogeneous insertion of virion antigen in the membrane of the target cell is sufficient for T cell cytotoxicity. The finding is incompatible with the concept that H-2K or H-2D gene products of the target cells must be altered in their primary structure (pretranslational effect of the virus infection) for T cell-mediated cytolysis to occur.
非感染性仙台病毒使H-2匹配的靶细胞易受仙台病毒免疫细胞毒性T淋巴细胞的裂解作用影响。这一观察结果表明，病毒粒子抗原在外源插入靶细胞膜中就足以引发T细胞细胞毒性。这一发现与以下概念不相符，即靶细胞的H-2K或H-2D基因产物的一级结构必须发生改变（病毒感染的翻译前效应）才能发生T细胞介导的细胞溶解。
