Another broken heart: loss of lamina-associated polypeptide 2alpha causes systolic dysfunction.
作者信息
Verstraeten Valerie L R M, Lammerding Jan
出版信息
Circ Res. 2010 Feb 5;106(2):234-7. doi: 10.1161/CIRCRESAHA.109.212001.
Abstract
摘要
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本文引用的文献
1
Lamina-associated polypeptide 2alpha loss impairs heart function and stress response in mice.层粘连蛋白相关多肽 2alpha 的缺失会损害小鼠的心脏功能和应激反应。
Circ Res. 2010 Feb 5;106(2):346-53. doi: 10.1161/CIRCRESAHA.109.205724. Epub 2009 Nov 19.
2
Attenuated hypertrophic response to pressure overload in a lamin A/C haploinsufficiency mouse. lamin A/C 杂合不足小鼠对压力超负荷的肥厚反应减弱。
J Mol Cell Cardiol. 2010 Jun;48(6):1290-7. doi: 10.1016/j.yjmcc.2009.10.024. Epub 2009 Nov 12.
3
Laminopathies and the long strange trip from basic cell biology to therapy.核纤层蛋白病以及从基础细胞生物学走向治疗的漫长而奇特之旅。
J Clin Invest. 2009 Jul;119(7):1825-36. doi: 10.1172/JCI37679. Epub 2009 Jul 1.
4
Lamin A/C deficiency as a cause of familial dilated cardiomyopathy.核纤层蛋白A/C缺乏作为家族性扩张型心肌病的一个病因
Curr Opin Cardiol. 2009 May;24(3):203-8. doi: 10.1097/HCO.0b013e32832a11c6.
5
Evidence for cardiomyocyte renewal in humans.人类心肌细胞更新的证据。
Science. 2009 Apr 3;324(5923):98-102. doi: 10.1126/science.1164680.
6
Inhibition of extracellular signal-regulated kinase signaling to prevent cardiomyopathy caused by mutation in the gene encoding A-type lamins.抑制细胞外信号调节激酶信号传导以预防由编码 A 型核纤层蛋白的基因突变引起的心肌病。
Hum Mol Genet. 2009 Jan 15;18(2):241-7. doi: 10.1093/hmg/ddn343. Epub 2008 Oct 16.
7
Loss of nucleoplasmic LAP2alpha-lamin A complexes causes erythroid and epidermal progenitor hyperproliferation.核质LAP2α-核纤层蛋白A复合物的缺失会导致红细胞和表皮祖细胞过度增殖。
Nat Cell Biol. 2008 Nov;10(11):1341-8. doi: 10.1038/ncb1793. Epub 2008 Oct 12.
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Identification of mutational hot spots in LMNA encoding lamin A/C in patients with familial dilated cardiomyopathy.家族性扩张型心肌病患者中编码核纤层蛋白A/C的LMNA基因的突变热点鉴定。
Basic Res Cardiol. 2009 Jan;104(1):90-9. doi: 10.1007/s00395-008-0748-6. Epub 2008 Sep 15.
9
Emerin and the nuclear lamina in muscle and cardiac disease.Emerin与肌肉和心脏疾病中的核纤层
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10
Lamin A/C mutation analysis in a cohort of 324 unrelated patients with idiopathic or familial dilated cardiomyopathy.对324名患有特发性或家族性扩张型心肌病的无血缘关系患者进行的核纤层蛋白A/C突变分析。
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