输入多样性对恒河猴肾细胞中猿猴病毒40持续感染建立的影响。
Effect of input multiplicity on the establishment of simian virus 40 persistent infections in rhesus monkey kidney cells.
作者信息
Norkin L C
出版信息
Infect Immun. 1977 Dec;18(3):868-71. doi: 10.1128/iai.18.3.868-871.1977.
Abstract
Monolayer cultures of LLC-MK2 rhesus monkey kidney cells become persistently infected with simian virus 40 after infection at input multiplicities of 100, 10, or 1 plaque-forming unit per cell. After 3 weeks, all cells of the cultures infected at a multiplicity of 1 plaque-forming unit per cell produced the simian virus 40 T antigen. In contrast, 8 to 11 weeks elapsed before all the cells in the cultures infected at a multiplicity of 100 plaque-forming units per cell produced T antigen. Defective interfering particles and interferon production were not evident during this time.
摘要
恒河猴肾LLC-MK2细胞单层培养物在以每细胞100、10或1个空斑形成单位的感染复数感染后，会持续感染猿猴病毒40。3周后，以每细胞1个空斑形成单位的感染复数感染的培养物中的所有细胞都产生了猿猴病毒40 T抗原。相比之下，以每细胞100个空斑形成单位的感染复数感染的培养物中，所有细胞产生T抗原则需要经过8至11周。在此期间，缺陷干扰颗粒和干扰素的产生并不明显。
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The vacuolating virus, S.V. 40.空泡病毒，即猴病毒40（SV40）
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