Heat shock protein 47 can be a new target molecule for intestinal fibrosis related to inflammatory bowel disease.

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作者信息

Honzawa Yusuke, Nakase Hiroshi, Takeda Yasuhiro, Nagata Kazuhiro, Chiba Tsutomu

出版信息

Inflamm Bowel Dis. 2010 Dec;16(12):2004-6. doi: 10.1002/ibd.21243.

DOI:10.1002/ibd.21243

PMID:20186932

Abstract

摘要

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Heat shock protein 47 can be a new target molecule for intestinal fibrosis related to inflammatory bowel disease.

Inflamm Bowel Dis. 2010 Dec;16(12):2004-6. doi: 10.1002/ibd.21243.

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引用本文的文献

Investigating the role of heat shock protein 47 in fibrosis in Crohn's disease.探讨热休克蛋白 47 在克罗恩病纤维化中的作用。

Sci Rep. 2022 Jun 29;12(1):10966. doi: 10.1038/s41598-022-15153-2.

Intestinal stenosis in Crohn's disease shows a generalized upregulation of genes involved in collagen metabolism and recognition that could serve as novel anti-fibrotic drug targets.克罗恩病中的肠道狭窄表现出参与胶原蛋白代谢和识别的基因普遍上调，这些基因可作为新型抗纤维化药物靶点。

Therap Adv Gastroenterol. 2020 Aug 29;13:1756284820952578. doi: 10.1177/1756284820952578. eCollection 2020.

Activation of Myofibroblast TRPA1 by Steroids and Pirfenidone Ameliorates Fibrosis in Experimental Crohn's Disease.类固醇和吡非尼酮激活肌成纤维细胞TRPA1可改善实验性克罗恩病的纤维化

Cell Mol Gastroenterol Hepatol. 2017 Dec 21;5(3):299-318. doi: 10.1016/j.jcmgh.2017.12.005. eCollection 2018 Mar.

Precision-cut rat, mouse, and human intestinal slices as novel models for the early-onset of intestinal fibrosis.精密切割的大鼠、小鼠和人类肠道切片作为肠道纤维化早期发病的新型模型。

Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12323.

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