Luz J G, Jaeger R G, de Araújo V C, de Rezende J R
Department of Oral and Maxillofacial Surgery, University of São Paulo, Brazil.
Int J Oral Maxillofac Surg. 1991 Feb;20(1):48-52. doi: 10.1016/s0901-5027(05)80697-6.
The effect of indirect trauma to the rat temporomandibular joint (TMJ) is analysed by means of an experimental model. The trauma, applied from an angle-glenoid fossa direction, produced injury of the TMJ. The histological data demonstrated that the impact could produce fractures of the glenoid fossa, but no hemarthrosis was observed. Trauma, both with or without fracture, caused proliferative changes in the TMJ. The glenoid fossa, the articular disk and the articular surface of the condyle were injured. Thickening of the articular surfaces had resulted in reduced joint space. Subsequently, remodelling changes in the condyle were found.
通过实验模型分析间接创伤对大鼠颞下颌关节(TMJ)的影响。从关节盂窝方向施加的创伤导致了颞下颌关节损伤。组织学数据表明,撞击可导致关节盂窝骨折,但未观察到关节积血。无论有无骨折,创伤均会引起颞下颌关节的增殖性变化。关节盂窝、关节盘和髁突的关节表面均受到损伤。关节表面增厚导致关节间隙减小。随后,在髁突发现了重塑变化。