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pms3是导致“农垦58S”原始光周期敏感雄性不育突变的基因座。

pms3 is the locus causing the original photoperiod-sensitive male sterility mutation of 'Nongken 58S'.

作者信息

Mei M, Chen L, Zhang Z, Li Z, Xu C, Zhang Q

机构信息

National Key Laboratory of Crop Genetic Improvement, Huazhong Agricultural University, 430070, Wuhan, China.

出版信息

Sci China C Life Sci. 1999 Jun;42(3):316-22. doi: 10.1007/BF03183609.

Abstract

Photoperiod-sensitive genic male sterile (PSGMS) rice is a very useful germplasm for hybrid rice development. It was first found as a spontaneous mutant in a japonica cultivar 'Nongken 58.pms3 on chromosome 12 was determined to be the locus where the original PSGMS mutation occurred, changing the normal cultivar Nongken 58 to PS-GMS Nongken 58S. Large amounts of RAPD and AFLP analyses were also conducted for the fine mapping of the pms3 genomic region, which resulted in 4 molecular markers linked to pms3. Although these markers somewhat increased the marker density of this region, the pms3 locus is still located in a marker-sparse region.

摘要

光周期敏感型雄性不育(PSGMS)水稻是杂交水稻培育中一种非常有用的种质资源。它最初是在粳稻品种“农垦58”中作为自发突变体被发现的。第12号染色体上的pms3被确定为最初PSGMS突变发生的位点,使正常品种农垦58变成了PS - GMS农垦58S。还进行了大量的RAPD和AFLP分析以对pms3基因组区域进行精细定位,结果得到了4个与pms3连锁的分子标记。尽管这些标记在一定程度上增加了该区域的标记密度,但pms3位点仍位于标记稀疏区域。

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