Gait Laboratory, Central Remedial Clinic, Clontard, Dublin 3, Ireland.
Clin Anat. 2010 Jul;23(5):586-92. doi: 10.1002/ca.20976.
Failure of the knee extensor mechanism is a potentially disastrous complication of diplegic cerebral palsy and if left undiagnosed may lead to a cessation of independent walking. The disruption of the extensor mechanism usually occurs through or distal to the patella. The aim of this article is to describe the knee kinematic pattern associated with such knee pathology. We also present a mathematical model of knee crouch that leads to this problem. In a retrospective review of patients with radiographically proven disruption, we compared the postfailure clinical and kinematic data to premorbid data. All patients included in this study had attended our clinical Gait Analysis Laboratory on two occasions. In the patients with disruption of the extensor mechanism, the kinematic pattern changed from crouch with shock absorption to one of increased crouch and loss of shock absorption. Clinical characteristics included knee flexion contracture and increased hamstring tightness. We demonstrate how the prefailure crouch position of the knee increases the flexor moment arm about the knee. We suggest that this knee crouch position during walking is the primary cause of pathology. Failure of the knee extensor mechanism is associated with a distinctive knee kinematic pattern. Regular gait analysis can help identify this pathology and enable treatment to be planned accordingly.
膝伸肌机制的失效是双瘫型脑瘫的一种潜在灾难性并发症,如果不加以诊断,可能会导致独立行走能力的丧失。伸肌机制的破坏通常发生在髌骨或其远端。本文旨在描述与这种膝关节病理相关的膝关节运动学模式。我们还提出了一个导致这种问题的膝屈曲畸形的数学模型。在对经影像学证实有伸肌机制破坏的患者进行回顾性研究中,我们将发病后的临床和运动学数据与发病前的数据进行了比较。本研究所有纳入的患者均在我院步态分析实验室接受了两次检查。在伸肌机制破坏的患者中,运动学模式从减震的膝屈曲畸形转变为膝屈曲畸形加重和减震丧失。临床特征包括膝关节屈曲挛缩和腘绳肌紧张度增加。我们证明了膝关节在发病前的屈曲位置如何增加了膝关节的屈肌力矩臂。我们认为,这种行走时的膝关节屈曲畸形是导致这种病理的主要原因。膝伸肌机制失效与独特的膝关节运动学模式相关。定期的步态分析可以帮助识别这种病理,并相应地制定治疗计划。