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镫骨手术后迟发性面瘫的磁共振成像表现。

Magnetic resonance imaging findings in delayed facial palsy after stapes surgery.

机构信息

Division of Head and Neck Surgery, University of California-Los Angeles School of Medicine, Los Angeles, California, U.S.A.

出版信息

Otol Neurotol. 2010 Sep;31(7):1153-6. doi: 10.1097/MAO.0b013e3181eb3259.

Abstract

OBJECTIVES

  1. To retrospectively review the pathophysiology and radiologic features of delayed facial palsy after stapedectomy. 2. To discuss the cause and management strategy of this unusual problem.

PATIENTS

Four hundred fifty stapedectomies performed between 2001 and 2007 by a single surgeon were retrospectively reviewed. Two patients in this series developed a delayed facial palsy postoperatively.

INTERVENTIONS

Magnetic resonance imaging (MRI) was performed on patients with delayed facial palsy after stapedectomy and was repeated as their clinical symptoms resolved. The patients were treated medically with oral corticosteroids and antiviral medications.

MAIN OUTCOME MEASURES

The clinical course and radiographic characteristics on MRI of patients with delayed facial palsy are presented.

RESULTS

Two of 450 patients who underwent stapedectomies (0.4%) during a 7-year period developed the rare complication of delayed facial palsy (postoperative Days 12 and 41, respectively). Both patients experienced periauricular pain and dysgeusia, followed by rapidly progressive complete facial nerve paralysis. The operated ear showed no sign of infection. MRI with gadolinium of the internal auditory canal demonstrated enhancement of the labyrinthine portion of the facial nerve as seen in Bell's palsy. Both patients were treated with oral corticosteroids and antiviral medications. Subsequent imaging revealed decreased enhancement with resolution of facial paralysis.

CONCLUSION

In the rare complication of delayed facial palsy after stapedectomy, MRI findings support the hypothesis that reactivation of a latent virus is the underlying cause. Therefore, delayed facial paralysis after stapedectomy should be treated similarly to Bell's palsy with steroid and antiviral medical therapy.

摘要

目的

  1. 回顾镫骨切除术后迟发性面瘫的病理生理学和放射学特征。2. 讨论这个不常见问题的原因和处理策略。

患者

回顾了 2001 年至 2007 年间由一位外科医生进行的 450 例镫骨切除术,该系列中有 2 例患者术后发生迟发性面瘫。

干预

对镫骨切除术后发生迟发性面瘫的患者进行磁共振成像(MRI)检查,并在其临床症状缓解时重复进行。患者接受了皮质类固醇和抗病毒药物的药物治疗。

主要观察指标

介绍了迟发性面瘫患者的临床过程和 MRI 影像学特征。

结果

在 7 年期间,450 例接受镫骨切除术的患者中有 2 例(0.4%)发生了罕见的迟发性面瘫并发症(分别在术后第 12 天和第 41 天)。两名患者均出现耳周疼痛和味觉障碍,随后迅速进展为完全性面瘫。手术耳无感染迹象。内听道 MRI 增强显示面神经迷路段增强,类似于贝尔面瘫。两名患者均接受了皮质类固醇和抗病毒药物治疗。随后的影像学检查显示面神经麻痹改善,增强减弱。

结论

在镫骨切除术后迟发性面瘫的罕见并发症中,MRI 结果支持病毒再激活是潜在原因的假说。因此,镫骨切除术后迟发性面瘫应类似于贝尔面瘫,采用类固醇和抗病毒药物治疗。

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