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巨噬细胞:炎症和纤维化的主要调节者。

Macrophages: master regulators of inflammation and fibrosis.

机构信息

Immunopathogenesis Section, Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA.

出版信息

Semin Liver Dis. 2010 Aug;30(3):245-57. doi: 10.1055/s-0030-1255354. Epub 2010 Jul 21.

Abstract

Macrophages are found in close proximity with collagen-producing myofibroblasts and indisputably play a key role in fibrosis. They produce profibrotic mediators that directly activate fibroblasts, including transforming growth factor-beta1 and platelet-derived growth factor, and control extracellular matrix turnover by regulating the balance of various matrix metalloproteinases and tissue inhibitors of matrix metalloproteinases. Macrophages also regulate fibrogenesis by secreting chemokines that recruit fibroblasts and other inflammatory cells. With their potential to act in both a pro- and antifibrotic capacity, as well as their ability to regulate the activation of resident and recruited myofibroblasts, macrophages and the factors they express are integrated into all stages of the fibrotic process. These various, and sometimes opposing, functions may be performed by distinct macrophage subpopulations, the identification of which is a growing focus of fibrosis research. Although collagen-secreting myofibroblasts once were thought of as the master "producers" of fibrosis, this review will illustrate how macrophages function as the master "regulators" of fibrosis.

摘要

巨噬细胞与产生胶原的肌成纤维细胞密切相邻,无疑在纤维化中发挥着关键作用。它们产生直接激活成纤维细胞的促纤维化介质,包括转化生长因子-β1 和血小板衍生生长因子,并通过调节各种基质金属蛋白酶和基质金属蛋白酶组织抑制剂的平衡来控制细胞外基质的周转。巨噬细胞还通过分泌趋化因子招募成纤维细胞和其他炎症细胞来调节纤维化。由于其具有在促纤维化和抗纤维化能力中发挥作用的潜力,以及调节固有和募集的肌成纤维细胞激活的能力,巨噬细胞及其表达的因子被整合到纤维化过程的所有阶段。这些不同的、有时是对立的功能可能由不同的巨噬细胞亚群来执行,其鉴定是纤维化研究的一个新兴焦点。尽管曾认为分泌胶原的肌成纤维细胞是纤维化的主要“产生者”,但本综述将说明巨噬细胞如何作为纤维化的主要“调节者”发挥作用。

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