Pandya S K, Nagpal R D, Desai A P, Purohit A V
J Neurosurg. 1978 Jun;48(6):1030-4. doi: 10.3171/jns.1978.48.6.1030.
A 24-year-old man with left sixth through twelfth cranial nerve palsies and severe, fluctuating, systemic arterial hypertension was found to have a large, vascular, catecholamine-forming glomus jugulare chemodectoma. After his electrolyte imbalance was corrected, left external carotid embolization (using Gelfoam) was carried out. The systemic blood pressure stabilized at around 160/100 mm Hg over the next 12 hours. Ten hours later the hitherto conscious patient developed acute arterial hypotension, lapsed into coma, and died. Autopsy showed tumor infarction, swelling, cerebellar tonsillar herniation, and medullary compression. An unusual complication of glomus tumor embolization is highlighted. The roles of preliminary decompressive surgery and urgent resuscitation by vasopressors are discussed.
一名24岁男性,患有左侧第六至第十二对脑神经麻痹以及严重的、波动的全身性动脉高血压,被发现患有一个巨大的、血管性的、形成儿茶酚胺的颈静脉球化学感受器瘤。在纠正其电解质失衡后,进行了左侧颈外动脉栓塞术(使用明胶海绵)。在接下来的12小时内,全身血压稳定在160/100 mmHg左右。10小时后,此前一直清醒的患者出现急性动脉低血压,陷入昏迷并死亡。尸检显示肿瘤梗死、肿胀、小脑扁桃体疝和延髓受压。强调了颈静脉球瘤栓塞术的一种不寻常并发症。讨论了初步减压手术和使用血管升压药进行紧急复苏的作用。