Uhrín P
Výskumný śtav zivocisnej výroby, Nitra.
Vet Med (Praha). 1990 Jul;35(7):403-9.
Post mortem glycolysis patterns were investigated in healthy pigs and in pigs with malignant hyperthermia syndrome (MHS). The beginning of glycolysis in the muscle of MHS-sensitive pigs is shifted, it is earlier than in the muscles of MHS-resistant animals. This earlier beginning is documented by lower glycogen values, higher lactate values, and also by the lower pH value as soon as 15 minutes after the slaughter of animals in this group. In the muscles of pigs with malignant hyperthermia syndrome the process of glycolysis is accelerated by the pre-slaughter stress. This is demonstrated first of all by a faster increase in the lactate content and by a faster decrease in the pH value in all periods of investigation. No discernible acceleration in the glycolysis pattern following the stress was observed in the MHS-resistant pigs. Glycogen depletion occurred in both groups of animals in all three types of muscles, the highest loss was recorded in white fibers.
对健康猪和患有恶性高热综合征(MHS)的猪的死后糖酵解模式进行了研究。MHS敏感猪肌肉中糖酵解的起始发生了变化,比MHS抗性动物的肌肉更早开始。糖原值较低、乳酸值较高,以及在该组动物屠宰后15分钟时pH值较低,都证明了这种更早的起始。在患有恶性高热综合征的猪的肌肉中,屠宰前应激加速了糖酵解过程。这首先表现为在所有研究阶段乳酸含量的更快增加和pH值的更快下降。在MHS抗性猪中未观察到应激后糖酵解模式有明显加速。两组动物的所有三种类型肌肉中均发生了糖原消耗,白色纤维中的损失最大。