Tynecka Z, Malm A, Zajac J
Department of Pharmaceutical Microbiology, Medical Academy, Lublin, Poland.
Acta Biochim Pol. 1990;37(1):121-4.
In the Cd2(+)-resistant Staphylococcus aureus 17810R which contains the plasmid-coded Cd2+ efflux system, accumulation of Cd2+ was highly reduced. Consequently, neither respiration nor ATP synthesis coupled to electron transfer were inhibited. The plasmidless S. aureus strain 17810S accumulated Cd2+ via the Mn2+ porter down the membrane potential (delta phi) which resulted in inhibition of respiration and of ATP synthesis.
在含有质粒编码的Cd2+外排系统的耐Cd2+金黄色葡萄球菌17810R中，Cd2+的积累显著减少。因此，呼吸作用和与电子传递偶联的ATP合成均未受到抑制。无质粒的金黄色葡萄球菌菌株17810S通过Mn2+转运体顺着膜电位（Δφ）积累Cd2+，这导致了呼吸作用和ATP合成受到抑制。
