Schäfer J, Pirsig W
Universität Ulm, HNO-Klinik, Sektion Rhinologie und Rhonchopathien, F.R.G.
Int J Pediatr Otorhinolaryngol. 1990 Dec;20(3):193-202. doi: 10.1016/0165-5876(90)90349-v.
Snoring sounds of 5 children with sleep-dependent compromised breathing were recorded. Underlying diseases were large adenoids, adeno-tonsillar hyperplasia, hemangioma of the tongue-base and Rubinstein-Taybi's syndrome associated with micrognathia. For comparison snoring sounds of a 42-year-old chronic snorer were analyzed. Time series of the power spectral density of the breath sounds were calculated as well as the mean power spectra of the recordings. The results show that the frequency spectrum reflects the pathomechanism responsible for the production of the abnormal breath sound. In adults, simple snoring is due in large part to vibrations of the soft palate. This can be identified in the frequency spectrum as a low-frequency component with a large number of harmonics. In children with enlarged adenoids and tonsils the soft palate is impeded in its movement which can be demonstrated in the frequency spectrum as a lack of low-frequency components and harmonics. Craniofacial anomalies are characterized by special spectral patterns.
记录了5名患有睡眠相关呼吸功能受损儿童的打鼾声。潜在疾病包括腺样体肥大、腺样体-扁桃体增生、舌根血管瘤以及与小颌畸形相关的鲁宾斯坦-泰比综合征。作为对照，分析了一名42岁慢性打鼾者的打鼾声。计算了呼吸音功率谱密度的时间序列以及录音的平均功率谱。结果表明，频谱反映了产生异常呼吸音的病理机制。在成年人中，单纯打鼾很大程度上是由于软腭振动引起的。这在频谱中可表现为具有大量谐波的低频成分。在腺样体和扁桃体肿大的儿童中，软腭运动受阻，这在频谱中表现为低频成分和谐波的缺失。颅面畸形具有特殊的频谱模式。
