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Airway hyperresponsiveness in asthma.

作者信息

Cockcroft D W

机构信息

Section of Respiratory Medicine, University of Saskatchewan College of Medicine, Saskatoon.

出版信息

Hosp Pract (Off Ed). 1990 Jan 15;25(1):111-8, 121, 125-9. doi: 10.1080/21548331.1990.11703899.

DOI:10.1080/21548331.1990.11703899
PMID:2104859
Abstract

Some asthma triggers exert their major action through cellular inflammatory mechanisms, with resultant airway hyperreactivity to both allergenic and nonallergenic stimuli. Other triggers are simply bronchospastic, so that they do not increase vulnerability. Anti-inflammatory treatments are more important than purely bronchodilator therapy is for long-term control of asthma.

摘要

相似文献

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Airway hyperresponsiveness in asthma.
Hosp Pract (Off Ed). 1990 Jan 15;25(1):111-8, 121, 125-9. doi: 10.1080/21548331.1990.11703899.
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引用本文的文献

1
Activities of superoxide dismutases and NADPH oxidase in neutrophils obtained from asthmatic and normal donors.从哮喘患者和正常供体获取的中性粒细胞中超氧化物歧化酶和NADPH氧化酶的活性。
Inflammation. 1993 Jun;17(3):361-70. doi: 10.1007/BF00918997.
2
Neutrophils from asthmatics exhibit diminished responsiveness to 2-chloroadenosine which is reversed by theophylline. Evidence for a cyclic-AMP-independent pathway on human neutrophils.哮喘患者的中性粒细胞对2-氯腺苷的反应性降低,而氨茶碱可逆转这种情况。关于人类中性粒细胞中一条不依赖环磷酸腺苷途径的证据。
Inflammation. 1992 Apr;16(2):101-16. doi: 10.1007/BF00918951.