Department of Orthopedic Surgery, Samsung Medical Center, Sungkyunkwan University School of Medicine, 50 Irwon-dong, Gangnam-gu, Seoul 135-710, Republic of Korea.
J Bone Joint Surg Am. 2010 Nov 17;92(16):2692-9. doi: 10.2106/JBJS.I.01367.
Several surgical approaches have been suggested for the treatment of posttraumatic elbow stiffness; however, the optimal approach to elbows with considerable loss of flexion has not been well described. We investigated the pathologic lesions causing posttraumatic loss of elbow flexion and analyzed the results of surgical release.
Forty-two patients with <100° of elbow flexion due to an extrinsic contracture following trauma underwent surgical release at a median of ten months after injury. To achieve maximum flexion, release of the posterior band of the medial collateral ligament was mandatory in all patients, and only four patients required additional anterior procedures. The ulnar nerve was transposed anteriorly in forty patients, including three who had had a previous transposition. To evaluate the results, we compared preoperative and postoperative elbow motion, Mayo Elbow Performance Index (MEPI) scores, and radiographs.
Intraoperatively, heterotopic ossification was observed in forty patients. It was located predominantly in the posteromedial aspect of the capsule. Heterotopic bone was more commonly found during surgery than it was identified preoperatively on radiographs. Mean flexion increased significantly from 89° preoperatively to 124° (range, 90° to 140°) at a mean of thirty-nine months postoperatively. The mean size of the flexion contracture decreased from 34° preoperatively to 9° (range, 0° to 30°) postoperatively. Overall, ≥120° of final flexion and a total arc of ≥100° were regained by 88% of the patients. The mean MEPI score improved significantly from 73 points preoperatively to 94 points (range, 72 to 100 points) postoperatively, with the result rated as excellent in thirty-two patients, good in eight, and fair in two. Two patients had clinical recurrence of heterotopic ossification associated with a failure to obtain an increase in flexion.
This study demonstrates that posttraumatic heterotopic ossification, particularly in the posteromedial aspect of the capsule, is closely associated with loss of elbow flexion. Satisfactory restoration of elbow flexion can be obtained in the majority of patients by surgical release of the posterior band of the medial collateral ligament and excision of heterotopic bone.
对于创伤后肘僵硬,已有多种手术方法被提出;然而,对于严重丧失屈曲的肘部,其最佳治疗方法尚未被很好地描述。我们研究了导致创伤后肘部丧失屈曲的病理损伤,并分析了手术松解的结果。
42 例患者因创伤后外在性挛缩而导致屈肘角度<100°,于受伤后平均 10 个月行手术松解。为获得最大屈曲度,所有患者均需行内侧副韧带后束松解,仅有 4 例患者需要行额外的前方松解。40 例患者行尺神经前置,其中 3 例为既往前置。为评估结果,我们比较了术前和术后的肘活动度、Mayo 肘功能评分(MEPI)和影像学结果。
术中发现 40 例患者存在异位骨化。其主要位于关节囊的后内侧。术中发现的异位骨化比术前影像学上更常见。平均屈曲度从术前的 89°显著增加至术后 39 个月时的 124°(范围为 90°至 140°)。术前挛缩角度为 34°,术后为 9°(范围为 0°至 30°)。总体而言,88%的患者最终获得了≥120°的屈曲度和≥100°的总活动度。术前 MEPI 评分为 73 分,术后为 94 分(范围为 72 至 100 分),评分显著提高,其中 32 例患者结果为优,8 例为良,2 例为可。2 例患者因未能获得屈曲度增加而出现异位骨化的临床复发。
本研究表明,创伤后异位骨化,特别是关节囊后内侧异位骨化,与肘部丧失屈曲密切相关。通过手术松解内侧副韧带后束和切除异位骨,大多数患者的肘屈曲度均可得到满意恢复。