Akter Nasima, Sobahan Muhammad Abdus, Hossain Mohammad Anowar, Uraji Misugi, Nakamura Yoshimasa, Mori Izumi C, Murata Yoshiyuki
Division of Bioscience, Graduate School of Natural Science and Technology, Okayama University, Okayama, Japan.
Biosci Biotechnol Biochem. 2010;74(12):2504-6. doi: 10.1271/bbb.100513. Epub 2010 Dec 7.
We examined the involvement of intracellular glutathione (GSH) in methyl jasmonate (MeJA) signaling. The chlorina1-1 (ch1-1) mutation decreased GSH in guard cells and narrowed the stomatal aperture. GSH monoethyl ester increased intracellular GSH, diminishing this phenotype. GSH did not affect MeJA-induced reactive oxygen species production or cytosolic Ca(2+) oscillation, suggesting that GSH modulates MeJA signaling downstream of production and oscillation.
我们研究了细胞内谷胱甘肽(GSH)在茉莉酸甲酯(MeJA)信号传导中的作用。chlorina1-1(ch1-1)突变降低了保卫细胞中的GSH含量,并使气孔孔径变窄。GSH单乙酯增加了细胞内的GSH,减轻了这种表型。GSH不影响MeJA诱导的活性氧产生或胞质Ca(2+)振荡,这表明GSH在产生和振荡的下游调节MeJA信号传导。