AS-1蓝噬菌体感染会抑制聚球藻属PCC 6301（一种蓝细菌）中光系统II的光合电子流。

AS-1 cyanophage infection inhibits the photosynthetic electron flow of photosystem II in Synechococcus sp. PCC 6301, a cyanobacterium.

作者信息

Teklemariam T A, Demeter S, Deák Z, Surányi G, Borbély G

机构信息

Institute of Plant Physiology, Biological Research Center, Szeged, Hungary.

出版信息

FEBS Lett. 1990 Sep 17;270(1-2):211-5. doi: 10.1016/0014-5793(90)81270-x.

DOI:10.1016/0014-5793(90)81270-x

PMID:2121523

Abstract

In Synechococcus sp. cells AS-1 cyanophage infection gradually inhibits the photosystem II mediated photosynthetic electron flow whereas the activity of photosystem I is apparently unaffected by the cyanophage infection. Transient fluorescence induction and flash-induced delayed luminescence decay studies revealed that the inhibition may occur at the level of the secondary acceptor, QB of photosystem II. In addition, the breakdown of D1-protein is inhibited, comparable to DCMU-induced protection of D1-protein turnover, in AS-1-infected cells.

摘要

在聚球藻属AS-1细胞中，蓝藻噬菌体感染会逐渐抑制光系统II介导的光合电子流，而光系统I的活性显然不受蓝藻噬菌体感染的影响。瞬态荧光诱导和闪光诱导延迟发光衰减研究表明，这种抑制可能发生在光系统II的次级受体QB水平。此外，在AS-1感染的细胞中，D1蛋白的降解受到抑制，这与二氯苯基二甲基脲（DCMU）诱导的对D1蛋白周转的保护作用相当。