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Mycobacterium tuberculosis evades macrophage defenses by inhibiting plasma membrane repair.
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Virulent Mycobacterium tuberculosis strains evade apoptosis of infected alveolar macrophages.
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Lipid mediators in innate immunity against tuberculosis: opposing roles of PGE2 and LXA4 in the induction of macrophage death.
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Cytotoxic granules and effector molecules from immune cells in tuberculosis: Mechanisms of host defense and therapeutic potential.
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Iron-restricted Mycobacterium tuberculosis exports pathogenicity factors packed in extracellular vesicles.
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Proapoptotic Bcl-2 inhibitor as potential host directed therapy for pulmonary tuberculosis.
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S-nitrosylation-triggered secretion of mycobacterial PknG leads to phosphorylation of SODD to prevent apoptosis of infected macrophages.
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本文引用的文献

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Human gene expression profiles of susceptibility and resistance in tuberculosis.
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Molecular mechanisms of necroptosis: an ordered cellular explosion.
Nat Rev Mol Cell Biol. 2010 Oct;11(10):700-14. doi: 10.1038/nrm2970. Epub 2010 Sep 8.
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The lta4h locus modulates susceptibility to mycobacterial infection in zebrafish and humans.
Cell. 2010 Mar 5;140(5):717-30. doi: 10.1016/j.cell.2010.02.013.
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Lipoxin A4 impairment of apoptotic signaling in macrophages: implication of the PI3K/Akt and the ERK/Nrf-2 defense pathways.
Cell Death Differ. 2010 Jul;17(7):1179-88. doi: 10.1038/cdd.2009.220. Epub 2010 Jan 22.
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Necroptosis as an alternative form of programmed cell death.
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Mycobacterium tuberculosis evades macrophage defenses by inhibiting plasma membrane repair.
Nat Immunol. 2009 Aug;10(8):899-906. doi: 10.1038/ni.1758. Epub 2009 Jun 28.

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