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颈脊髓损伤后低钠血症 1 例。

A case of hyponatremia after cervical spinal cord injury.

机构信息

Department of Endocrinology and Metabolism, Hirosaki University Graduate School of Medicine, Hirosaki, Japan.

出版信息

Endocr J. 2011;58(5):369-72. doi: 10.1507/endocrj.k11e-038. Epub 2011 Mar 25.

DOI:10.1507/endocrj.k11e-038
PMID:21441702
Abstract

Hyponatremia is known as a complication of neurosurgical conditions or brain. The prevalence of hyponatremia in acute spinal cord injury has been known to be much higher than in the general medical or surgical patient population. We report here a case of hyponatremia, which occurred 10 days after cervical spinal cord injury. The data on sodium levels were improved under the tapered infusion volume and water intake restriction. Treatment for this case is the same as that for syndrome of inappropriate antidiuretic hormone, which have suggested that reduced extracellular volume is important to improve the hyponatremia after traumatic spinal cord injury. Uncontrolled hyponatremia may lead to lethargy, seizures, coma, cardiac arrhythmia and death. Therefore, the complication of hyponatremia should be paid attention after cervical spinal cord injury. A careful monitoring of sodium levels after the injury is required in the cases of cervical spinal cord injury.

摘要

低钠血症是一种与神经外科疾病或脑相关的并发症。急性脊髓损伤患者的低钠血症患病率明显高于一般内科或外科患者人群。我们在此报告一例发生于颈脊髓损伤 10 天后的低钠血症病例。通过逐渐减少输液量和限制水分摄入,血钠数据得到了改善。这种情况下的治疗与抗利尿激素不适当分泌综合征相同,这表明减少细胞外液体积对于改善创伤性脊髓损伤后的低钠血症很重要。未经控制的低钠血症可能导致昏睡、癫痫发作、昏迷、心律失常和死亡。因此,颈脊髓损伤后应注意低钠血症等并发症。在颈脊髓损伤的情况下,需要在损伤后仔细监测钠水平。

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