• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Dissecting the role of SALL4, a newly identified stem cell factor, in chronic myelogenous leukemia.

作者信息

Lu J, Ma Y, Kong N, Alipio Z, Gao C, Krause D S, Silberstein L E, Chai L

出版信息

Leukemia. 2011 Jul;25(7):1211-3. doi: 10.1038/leu.2011.65. Epub 2011 Apr 5.

DOI:10.1038/leu.2011.65
PMID:21468036
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3675449/
Abstract
摘要

相似文献

1
Dissecting the role of SALL4, a newly identified stem cell factor, in chronic myelogenous leukemia.剖析新发现的干细胞因子SALL4在慢性粒细胞白血病中的作用。
Leukemia. 2011 Jul;25(7):1211-3. doi: 10.1038/leu.2011.65. Epub 2011 Apr 5.
2
Molecular biology of chronic myeloid leukemia.慢性髓系白血病的分子生物学
Int J Hematol. 2001 Apr;73(3):308-22. doi: 10.1007/BF02981955.
3
Hematopoietic stem cells, leukemic stem cells and chronic myelogenous leukemia.造血干细胞、白血病干细胞与慢性粒细胞白血病
Cell Cycle. 2005 Feb;4(2):266-8. Epub 2005 Feb 20.
4
Abnormality of c-kit oncoprotein in certain patients with chronic myelogenous leukemia--potential clinical significance.某些慢性粒细胞白血病患者中c-kit癌蛋白的异常——潜在的临床意义。
Leukemia. 2002 Feb;16(2):170-7. doi: 10.1038/sj.leu.2402341.
5
Bmi1 reprograms CML B-lymphoid progenitors to become B-ALL-initiating cells.BMI1 将 CML B 淋巴样祖细胞重编程为 B-ALL 起始细胞。
Blood. 2012 Jan 12;119(2):494-502. doi: 10.1182/blood-2011-06-359232. Epub 2011 Nov 18.
6
Molecular basis for stem-cell self-renewal.干细胞自我更新的分子基础。
Hematol J. 2004;5 Suppl 3:S118-21. doi: 10.1038/sj.thj.6200436.
7
BMI-1 expression is enhanced through transcriptional and posttranscriptional regulation during the progression of chronic myeloid leukemia.在慢性髓性白血病进展过程中,BMI-1表达通过转录和转录后调控得以增强。
Ann Hematol. 2009 Apr;88(4):333-40. doi: 10.1007/s00277-008-0603-8. Epub 2008 Sep 10.
8
The Bmi-1 polycomb protein antagonizes the (-)-epigallocatechin-3-gallate-dependent suppression of skin cancer cell survival.BMI-1 多梳蛋白拮抗(-)-表没食子儿茶素没食子酸酯依赖性抑制皮肤癌细胞存活。
Carcinogenesis. 2010 Mar;31(3):496-503. doi: 10.1093/carcin/bgp314. Epub 2009 Dec 16.
9
The expression pattern of erythrocyte/megakaryocyte-related transcription factors GATA-1 and the stem cell leukemia gene correlates with hematopoietic differentiation and is associated with outcome of acute myeloid leukemia.红细胞/巨核细胞相关转录因子GATA-1和干细胞白血病基因的表达模式与造血分化相关,并与急性髓系白血病的预后相关。
Blood. 1995 Oct 15;86(8):3173-80.
10
Molecular pathogenesis of chronic myeloid leukemia: implications for new therapeutic strategies.慢性髓性白血病的分子发病机制:对新治疗策略的启示
Ann Hematol. 1999 Feb;78(2):49-64. doi: 10.1007/s002770050473.

引用本文的文献

1
Cancer stem cells: advances in knowledge and implications for cancer therapy.癌症干细胞:知识进展及其对癌症治疗的影响。
Signal Transduct Target Ther. 2024 Jul 5;9(1):170. doi: 10.1038/s41392-024-01851-y.
2
The new advance of SALL4 in cancer: Function, regulation, and implication.SALL4 在癌症中的新进展:功能、调控及意义。
J Clin Lab Anal. 2023 May;37(9-10):e24927. doi: 10.1002/jcla.24927. Epub 2023 Jun 20.
3
SALL4 correlates with proliferation, metastasis, and poor prognosis in prostate cancer by affecting MAPK pathway.

本文引用的文献

1
Bmi-1 is a target gene for SALL4 in hematopoietic and leukemic cells.Bmi-1是造血细胞和白血病细胞中SALL4的靶基因。
Proc Natl Acad Sci U S A. 2007 Jun 19;104(25):10494-9. doi: 10.1073/pnas.0704001104. Epub 2007 Jun 8.
2
The polycomb group BMI1 gene is a molecular marker for predicting prognosis of chronic myeloid leukemia.多梳蛋白家族BMI1基因是预测慢性髓性白血病预后的分子标志物。
Blood. 2007 Jul 1;110(1):380-3. doi: 10.1182/blood-2006-12-065599. Epub 2007 Mar 14.
3
Sall4 modulates embryonic stem cell pluripotency and early embryonic development by the transcriptional regulation of Pou5f1.
SALL4 通过影响 MAPK 通路与前列腺癌的增殖、转移和不良预后相关。
Cancer Med. 2023 Jun;12(12):13471-13485. doi: 10.1002/cam4.5998. Epub 2023 Apr 29.
4
SALL4: An Intriguing Therapeutic Target in Cancer Treatment.SALL4:癌症治疗中一个引人关注的治疗靶点。
Cells. 2022 Aug 20;11(16):2601. doi: 10.3390/cells11162601.
5
Demethylation and Up-Regulation of an Oncogene after Hypomethylating Therapy.去甲基化治疗后癌基因的去甲基化和上调。
N Engl J Med. 2022 May 26;386(21):1998-2010. doi: 10.1056/NEJMoa2119771.
6
SALL4 Oncogenic Function in Cancers: Mechanisms and Therapeutic Relevance.SALL4 在癌症中的致癌功能:机制和治疗相关性。
Int J Mol Sci. 2022 Feb 12;23(4):2053. doi: 10.3390/ijms23042053.
7
Identification of Clinical Relevant Molecular Subtypes of Pheochromocytoma.鉴定嗜铬细胞瘤的临床相关分子亚型。
Front Endocrinol (Lausanne). 2021 Jun 21;12:605797. doi: 10.3389/fendo.2021.605797. eCollection 2021.
8
Spalt-like transcription factor 4 expression in oral epithelial dysplasia and oral squamous cell carcinoma: An immunohistochemical appraisal.Spalt样转录因子4在口腔上皮发育异常及口腔鳞状细胞癌中的表达:一项免疫组织化学评估
J Carcinog. 2020 Nov 25;19:12. doi: 10.4103/jcar.JCar_13_20. eCollection 2020.
9
Effect of SALL4 on the Proliferation, Invasion and Apoptosis of Breast Cancer Cells.SALL4 对乳腺癌细胞增殖、侵袭和凋亡的影响。
Technol Cancer Res Treat. 2020 Jan-Dec;19:1533033820980074. doi: 10.1177/1533033820980074.
10
The transcription factor Spalt and human homologue SALL4 induce cell invasion via the dMyc-JNK pathway in .转录因子 Spalt 和人同源物 SALL4 通过 dMyc-JNK 通路诱导 细胞侵袭。
Biol Open. 2020 Mar 24;9(3):bio048850. doi: 10.1242/bio.048850.
Sall4 通过对Pou5f1的转录调控来调节胚胎干细胞的多能性和早期胚胎发育。
Nat Cell Biol. 2006 Oct;8(10):1114-23. doi: 10.1038/ncb1481. Epub 2006 Sep 17.
4
SALL4, a novel oncogene, is constitutively expressed in human acute myeloid leukemia (AML) and induces AML in transgenic mice.SALL4是一种新型致癌基因,在人类急性髓系白血病(AML)中持续表达,并在转基因小鼠中诱发AML。
Blood. 2006 Oct 15;108(8):2726-35. doi: 10.1182/blood-2006-02-001594. Epub 2006 Jun 8.
5
Granulocyte-macrophage progenitors as candidate leukemic stem cells in blast-crisis CML.粒-巨噬细胞祖细胞作为急变期慢性粒细胞白血病中候选白血病干细胞
N Engl J Med. 2004 Aug 12;351(7):657-67. doi: 10.1056/NEJMoa040258.
6
Self-renewal of hematopoietic and leukemic stem cells: a central role for the Polycomb-group gene Bmi-1.造血干细胞和白血病干细胞的自我更新:多梳蛋白家族基因Bmi-1的核心作用。
Trends Immunol. 2003 Oct;24(10):522-4. doi: 10.1016/s1471-4906(03)00241-2.
7
Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells.Bmi-1是维持成年自我更新造血干细胞所必需的。
Nature. 2003 May 15;423(6937):302-5. doi: 10.1038/nature01587. Epub 2003 Apr 20.
8
The P190, P210, and P230 forms of the BCR/ABL oncogene induce a similar chronic myeloid leukemia-like syndrome in mice but have different lymphoid leukemogenic activity.BCR/ABL癌基因的P190、P210和P230形式在小鼠中诱导出类似慢性髓性白血病的综合征,但具有不同的淋巴细胞白血病致瘤活性。
J Exp Med. 1999 May 3;189(9):1399-412. doi: 10.1084/jem.189.9.1399.