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本文引用的文献

1
Mesenchymal stem cells for acute lung injury: preclinical evidence.间充质干细胞治疗急性肺损伤：临床前证据。
Crit Care Med. 2010 Oct;38(10 Suppl):S569-73. doi: 10.1097/CCM.0b013e3181f1ff1d.
2
The science of translating research into practice in intensive care.重症监护中研究成果转化为实际应用的科学。
Am J Respir Crit Care Med. 2010 Dec 15;182(12):1463-4. doi: 10.1164/rccm.201008-1255ED.
3
Moving the science of quality improvement in critical care medicine forward.推动重症医学质量改进科学向前发展。
Am J Respir Crit Care Med. 2010 Dec 15;182(12):1461-2. doi: 10.1164/rccm.201009-1483ED.
4
Metabolomics of sepsis-induced acute lung injury: a new approach for biomarkers.脓毒症诱导的急性肺损伤的代谢组学：生物标志物的新方法
Am J Physiol Lung Cell Mol Physiol. 2011 Jan;300(1):L1-3. doi: 10.1152/ajplung.00375.2010. Epub 2010 Nov 5.
5
Mechanism of transfusion-related acute lung injury induced by HLA class II antibodies.HLA Ⅱ类抗体引起的输血相关性急性肺损伤的机制。
Blood. 2011 Jan 13;117(2):669-77. doi: 10.1182/blood-2010-05-286146. Epub 2010 Oct 28.
6
Epithelial cell death is an important contributor to oxidant-mediated acute lung injury.上皮细胞死亡是氧化剂介导的急性肺损伤的重要原因。
Am J Respir Crit Care Med. 2011 Apr 15;183(8):1043-54. doi: 10.1164/rccm.201002-0181OC. Epub 2010 Oct 19.
7
Metabolic consequences of sepsis-induced acute lung injury revealed by plasma ¹H-nuclear magnetic resonance quantitative metabolomics and computational analysis.血浆 ¹H 核磁共振定量代谢组学和计算分析揭示脓毒症诱导的急性肺损伤的代谢后果。
Am J Physiol Lung Cell Mol Physiol. 2011 Jan;300(1):L4-L11. doi: 10.1152/ajplung.00231.2010. Epub 2010 Oct 1.
8
Abl tyrosine kinase phosphorylates nonmuscle Myosin light chain kinase to regulate endothelial barrier function. Abl 酪氨酸激酶磷酸化非肌肉肌球蛋白轻链激酶以调节内皮屏障功能。
Mol Biol Cell. 2010 Nov 15;21(22):4042-56. doi: 10.1091/mbc.E09-10-0876. Epub 2010 Sep 22.
9
Neuromuscular blockers in early acute respiratory distress syndrome.早期急性呼吸窘迫综合征中的神经肌肉阻滞剂。
N Engl J Med. 2010 Sep 16;363(12):1107-16. doi: 10.1056/NEJMoa1005372.
10
Peroxisome proliferator-activated receptors gamma coactivator-1alpha: master regulator of mitochondrial biogenesis and survival during critical illness?过氧化物酶体增殖物激活受体γ辅激活因子-1α：危重症期间线粒体生物合成与存活的主要调节因子？
Am J Respir Crit Care Med. 2010 Sep 15;182(6):726-8. doi: 10.1164/rccm.201005-0695ED.

Update in acute lung injury and critical care 2010.

作者信息

Vadász István, Sznajder Jacob I

机构信息

Department of Internal Medicine, University of Giessen Lung Center, Justus Liebig University, Klinikstrasse 36, 35392 Giessen, Germany.

出版信息

Am J Respir Crit Care Med. 2011 May 1;183(9):1147-52. doi: 10.1164/rccm.201102-0327UP.

DOI:10.1164/rccm.201102-0327UP
PMID:21531954
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3114050/
Abstract
摘要