Assmus H, Antoniadis G, Bischoff C, Hoffmann R, Martini A-K, Preissler P, Scheglmann K, Schwerdtfeger K, Wessels K D, Wüstner-Hofmann M
Praxis für, periphere Neurochirurgie, Dossenheim, Germany.
Cent Eur Neurosurg. 2011 May;72(2):90-8. doi: 10.1055/s-0031-1271800. Epub 2011 May 4.
Cubital tunnel syndrome (CuTS) is the second most common peripheral nerve compression syndrome. In German-speaking countries, cubital tunnel syndrome is often referred to as sulcus ulnaris syndrome (retrocondylar groove syndrome). This term is anatomically incorrect, since the site of compression comprises not only the retrocondylar groove but the cubital tunnel, which consists of 3 parts: the retrocondylar groove, partially covered by the cubital tunnel retinaculum (lig. arcuatum or Osborne ligament), the humeroulnar arcade, and the deep flexor/pronator aponeurosis. According to Sunderland , cubital tunnel syndrome can be differentiated into a primary form (including anterior subluxation of the ulnar nerve and compression secondary to the presence of an anconeus epitrochlearis muscle) and a secondary form caused by deformation or other processes of the elbow joint. The clinical diagnosis is usually confirmed by nerve conduction studies. Recently, the use of ultrasound and MRI have become useful diagnostic tools by showing morphological changes in the nerve within the cubital tunnel. A differential diagnosis is essential in atypical cases, and should include such conditions as C8 radiculopathy, Pancoast tumor, and pressure palsy. Conservative treatment (avoiding exposure to external noxes and applying of night splints) may be considered in the early stages of cubital tunnel syndrome. When nonoperative treatment fails, or in patients who present with more advanced clinical findings, such as motor weakness, muscle atrophy, or fixed sensory changes, surgical treatment should be recommended. According to actual randomized controlled studies, the treatment of choice in primary cubital tunnel syndrome is simple in situ decompression, which has to be extended at least 5-6 cm distal to the medial epicondyle and can be performed by an open or endoscopic technique, both under local anesthesia. Simple decompression is also the therapy of choice in uncomplicated ulnar luxation and in most post-traumatic cases and other secondary forms. When the luxation is painful, or when the ulnar nerve actually "snaps" back and forth over the medial epicondyle of the humerus, subcutaneous anterior transposition may be performed. In cases of severe bone or tissue changes of the elbow (especially with cubitus valgus), the anterior transposition of the ulnar nerve may again be indicated. In cases of scarring, submuscular transposition may be preferred as it provides a healthy vascular bed for the nerve as well as soft tissue protection. Risks resulting from transposition include compromise in blood flow to the nerve as well as kinking of the nerve caused by insufficient proximal or distal mobilization. In these cases, revision surgery is necessary. Epicondylectomy is not common, at least in Germany. Recurrence of compression on the ulnar nerve at the elbow may occur. This review is based on the German Guideline "Diagnose und Therapie des Kubitaltunnelsyndroms" ( www.leitlinien.net ).
肘管综合征(CuTS)是第二常见的周围神经卡压综合征。在德语国家,肘管综合征常被称为尺神经沟综合征(髁后沟综合征)。这个术语在解剖学上是不正确的,因为压迫部位不仅包括髁后沟,还包括肘管,肘管由三部分组成:髁后沟,部分被肘管支持带(弓状韧带或奥斯本韧带)覆盖;肱尺肌弓;以及深层屈肌/旋前肌腱膜。根据桑德兰的分类,肘管综合征可分为原发性(包括尺神经前脱位以及因肱肌滑车肌存在导致的压迫)和由肘关节变形或其他病变引起的继发性。临床诊断通常通过神经传导研究来确诊。近来,超声和磁共振成像(MRI)通过显示肘管内神经的形态变化成为有用的诊断工具。在非典型病例中,鉴别诊断至关重要,并应包括如C8神经根病、潘科斯特肿瘤和压迫性麻痹等情况。肘管综合征早期可考虑保守治疗(避免接触外部有害因素并应用夜间夹板)。当非手术治疗失败,或患者出现更严重的临床表现,如运动无力、肌肉萎缩或固定的感觉改变时,应建议手术治疗。根据实际随机对照研究,原发性肘管综合征的首选治疗方法是简单的原位减压,减压范围必须至少在内上髁远端5 - 6厘米,可通过开放或内镜技术在局部麻醉下进行。简单减压也是单纯尺神经脱位、大多数创伤后病例及其他继发性肘管综合征的首选治疗方法。当脱位引起疼痛,或尺神经实际上在肱骨内上髁上来回“弹响”时,可进行皮下前置术。在肘部严重的骨骼或组织改变(尤其是伴有肘外翻)的情况下,可能再次需要进行尺神经前置术。在瘢痕形成的情况下,肌下前置术可能更受青睐,因为它为神经提供了健康的血管床以及软组织保护。前置术带来的风险包括神经血流受损以及因近端或远端活动不足导致的神经扭结。在这些情况下,需要进行翻修手术。肱骨内上髁切除术并不常见,至少在德国如此。肘部尺神经受压可能会复发。本综述基于德国指南《肘管综合征的诊断与治疗》(www.leitlinien.net)。