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1 型 Bland Sutton 结肠闭锁合并胎儿水肿的早产儿。

Type 1 Bland Sutton colonic atresia complicated by fetalis hydrops in a premature neonate.

机构信息

University of Glasgow School of Medicine, Wolfson Medical School Building, University Avenue, Glasgow, G12 8QQ, Scotland, UK.

出版信息

Scott Med J. 2011 May;56(2):120. doi: 10.1258/smj.2011.011102.

Abstract

Colonic atresia (CA) is an unusual cause of neonatal intestinal obstruction where a section of the colon has failed to form, leading to blockage or absence. A premature baby was delivered at 32 weeks of gestation via caesarian section following fetal distress. She was grossly oedematous and diagnosed with severe fetalis hydrops secondary to anaemia. She was resuscitated and stabilized. On the sixth day of life, the neonate's abdomen became severely distended with billous vomiting and failure to pass meconium. We suspected intestinal obstruction and performed an omnipaque enema which revealed dilated small bowel loops and a bowel atresia. Exploratory laparotomy confirmed a Type 1 Bland Sutton CA with mucosal web. An end colostomy was successfully performed and uneventful. In our case report, we describe a rare occurrence of postnatally diagnosed CA, complicated by fetalis hydrops and anaemia.

摘要

先天性结肠闭锁(CA)是新生儿肠梗阻的一种罕见病因,其特征为结肠的某一节段未能形成,导致肠腔阻塞或缺失。一名 32 周早产儿因胎儿窘迫行剖宫产分娩,患儿出生时全身水肿明显,诊断为严重胎儿水肿继发于贫血。患儿经过复苏和稳定治疗。生后第 6 天,患儿腹部显著膨隆,胆汁性呕吐,胎粪未解。我们怀疑存在肠梗阻,并进行了泛影葡胺灌肠检查,结果显示小肠扩张,存在肠闭锁。剖腹探查证实为 Bland Sutton Ⅰ型 CA,伴有黏膜隔膜。成功进行了末端结肠造口术,过程顺利。在本病例报告中,我们描述了一种罕见的产后诊断的 CA,其并发胎儿水肿和贫血。

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