Bonetskiĭ A A, Fedorov V I
Fiziol Zh SSSR Im I M Sechenova. 1990 Apr;76(4):486-91.
I. v. administration of angiotensin II (0.1, 0.3 and 1.0 microgram/kg/min for 5 min) raised the plasma norepinephrine level in the right atrium and carotid artery in a reverse dose-dependent manner. The veno-arterial difference decreased dose-dependently to 17.4, 7.2 and 0.1 per cent, resp., versus 21.8 per cent in the control group. The plasma norepinephrine level was decreased and the veno-arterial difference was similar to basal level at 1.0 microgram/kg/min dose of angiotensin II for 40 min. Angiotensin II seems to affect the lung uptake and inactivation of norepinephrine in phasic manner.
静脉注射血管紧张素II（0.1、0.3和1.0微克/千克/分钟，持续5分钟）以剂量反向依赖的方式提高了右心房和颈动脉中的血浆去甲肾上腺素水平。静脉-动脉差异呈剂量依赖性下降，分别降至17.4%、7.2%和0.1%，而对照组为21.8%。在血管紧张素II剂量为1.0微克/千克/分钟、持续40分钟时，血浆去甲肾上腺素水平降低，静脉-动脉差异与基础水平相似。血管紧张素II似乎以阶段性方式影响肺对去甲肾上腺素的摄取和失活。
