The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase.
作者信息
Shears S B, Dawson A P, Loomis-Husselbee J W, Cullen P J
出版信息
Biochem J. 1990 Sep 15;270(3):837. doi: 10.1042/bj2700837a.
Abstract
摘要
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The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase.
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本文引用的文献
1
Repetitive transient rises in cytoplasmic free calcium in hormone-stimulated hepatocytes.
Nature. 1986;319(6054):600-2. doi: 10.1038/319600a0.
2
The mechanism of action of GTP on Ca2+ efflux from rat liver microsomal vesicles.
Biochem J. 1987 May 15;244(1):87-92. doi: 10.1042/bj2440087.
3
Spatial and temporal aspects of cell signalling.
Philos Trans R Soc Lond B Biol Sci. 1988 Jul 26;320(1199):325-43. doi: 10.1098/rstb.1988.0080.
4
Inositol phosphates: proliferation, metabolism and function.
Philos Trans R Soc Lond B Biol Sci. 1988 Jul 26;320(1199):281-98. doi: 10.1098/rstb.1988.0077.
5
Aluminum, chemical physiology, and Alzheimer's disease.
Lancet. 1988 Oct 29;2(8618):1008-10. doi: 10.1016/s0140-6736(88)90754-4.
6
7
Inhibition of transducin activation and guanosine triphosphatase activity by aluminum ion.
J Biol Chem. 1989 Jan 5;264(1):243-50.
8
Aluminium perturbs oscillatory phosphoinositide-mediated calcium signalling in hormone-stimulated hepatocytes.
Biochem J. 1990 Jul 15;269(2):547-50. doi: 10.1042/bj2690547.
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