The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase.
作者信息
Shears S B, Dawson A P, Loomis-Husselbee J W, Cullen P J
出版信息
Biochem J. 1990 Sep 15;270(3):837. doi: 10.1042/bj2700837a.
Abstract
摘要
相似文献
1
The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase.铝对肝细胞中受体产生的钙瞬变的干扰并非由于Ins(1,4,5)P3刺激的Ca2+释放或5-磷酸酶和3-激酶对Ins(1,4,5)P3代谢的影响。
Biochem J. 1990 Sep 15;270(3):837. doi: 10.1042/bj2700837a.
2
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引用本文的文献
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Aluminum interaction with phosphoinositide-associated signal transduction.铝与磷酸肌醇相关信号转导的相互作用。
Arch Toxicol. 1994;68(1):1-7. doi: 10.1007/s002040050023.
2
Effects of aluminium on the hepatic inositol polyphosphate phosphatase.铝对肝脏肌醇多磷酸磷酸酶的影响。
Biochem J. 1995 Jan 15;305 ( Pt 2)(Pt 2):557-61. doi: 10.1042/bj3050557.
3
Electroporation can cause artefacts due to solubilization of cations from the electrode plates. Aluminum ions enhance conversion of inositol 1,3,4,5-tetrakisphosphate into inositol 1,4,5-trisphosphate in electroporated L1210 cells.电穿孔可能会因电极板上阳离子的溶解而导致假象。铝离子可增强电穿孔处理的L1210细胞中肌醇1,3,4,5-四磷酸向肌醇1,4,5-三磷酸的转化。
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本文引用的文献
1
Repetitive transient rises in cytoplasmic free calcium in hormone-stimulated hepatocytes.激素刺激的肝细胞中细胞质游离钙的重复性短暂升高。
Nature. 1986;319(6054):600-2. doi: 10.1038/319600a0.
2
The mechanism of action of GTP on Ca2+ efflux from rat liver microsomal vesicles.鸟苷三磷酸（GTP）对大鼠肝微粒体囊泡中钙离子外流的作用机制。
Biochem J. 1987 May 15;244(1):87-92. doi: 10.1042/bj2440087.
3
Spatial and temporal aspects of cell signalling.细胞信号传导的空间和时间方面。
Philos Trans R Soc Lond B Biol Sci. 1988 Jul 26;320(1199):325-43. doi: 10.1098/rstb.1988.0080.
4
Inositol phosphates: proliferation, metabolism and function.肌醇磷酸：增殖、代谢与功能
Philos Trans R Soc Lond B Biol Sci. 1988 Jul 26;320(1199):281-98. doi: 10.1098/rstb.1988.0077.
5
Aluminum, chemical physiology, and Alzheimer's disease.铝、化学生理学与阿尔茨海默病。
Lancet. 1988 Oct 29;2(8618):1008-10. doi: 10.1016/s0140-6736(88)90754-4.
6
The pathway of myo-inositol 1,3,4-trisphosphate phosphorylation in liver. Identification of myo-inositol 1,3,4-trisphosphate 6-kinase, myo-inositol 1,3,4-trisphosphate 5-kinase, and myo-inositol 1,3,4,6-tetrakisphosphate 5-kinase.肝脏中肌醇1,3,4-三磷酸的磷酸化途径。肌醇1,3,4-三磷酸6-激酶、肌醇1,3,4-三磷酸5-激酶和肌醇1,3,4,6-四磷酸5-激酶的鉴定。
J Biol Chem. 1989 Nov 25;264(33):19879-86.
7
Inhibition of transducin activation and guanosine triphosphatase activity by aluminum ion.铝离子对转导素激活和鸟苷三磷酸酶活性的抑制作用。
J Biol Chem. 1989 Jan 5;264(1):243-50.
8
Aluminium perturbs oscillatory phosphoinositide-mediated calcium signalling in hormone-stimulated hepatocytes.铝会干扰激素刺激的肝细胞中振荡性磷酸肌醇介导的钙信号传导。
Biochem J. 1990 Jul 15;269(2):547-50. doi: 10.1042/bj2690547.
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