Abdel-Aziz Mosaad, Ibrahim Neamat, Ahmed Abeer, El-Hamamsy Mostafa, Abdel-Khalik Mohamed I, El-Hoshy Hassan
Department of Otolaryngology, Faculty of Medicine, Cairo University, Egypt.
Int J Pediatr Otorhinolaryngol. 2011 Sep;75(9):1127-31. doi: 10.1016/j.ijporl.2011.06.003. Epub 2011 Jul 7.
Although adenotonsillar hypertrophy has been reported to be the commonest cause of pediatric obstructive sleep apnea (OSA), enlargement of the lingual tonsils is increasingly being recognized as a cause, even after adenotonsillectomy. The aim of our study was to elucidate the lingual tonsils hypertrophy as a cause of pediatric OSA and also to evaluate the efficacy of lingual tonsillectomy in relieving symptoms of the disease considering the peri-operative problems and management.
Sixteen children with lingual tonsils hypertrophy after adenotonsillectomy were included in the study. Computerized tomography (CT) and/or magnetic resonance imaging (MRI) were used for detection of the lesions. They underwent lingual tonsillectomy with special anesthetic care, flexible laryngoscopy and polysomnography were done pre- and post-operatively. Follow up of the patients was carried out for at least 1 year.
Three cases developed post-operative airway obstruction that is caused by tongue base edema. Complete improvement of snoring and apnea was achieved in 10 cases. Despite complete ablation of lingual tonsils, persistent snoring was detected in six cases, while apnea was detected in two cases. Down's syndrome, mucopolysaccharidoses, and obesity may be underlying factors for persistent symptoms.
Lingual tonsils hypertrophy could be the cause of obstructive sleep apnea in children after adenotonsillectomy, lingual tonsillectomy is an effective treatment for these cases, however peri-operative airway problems should be expected and can be managed safely. Persistent symptoms after lingual tonsillectomy may be due to the presence of co-morbidities such as cranio-facial deformities, obesity, and/or mucopolysaccharidoses.
尽管腺样体扁桃体肥大被报道为小儿阻塞性睡眠呼吸暂停(OSA)最常见的原因,但舌扁桃体肥大作为病因正日益受到认可,即便在腺样体扁桃体切除术后亦是如此。我们研究的目的是阐明舌扁桃体肥大作为小儿OSA病因的情况,并考虑围手术期问题及处理方法,评估舌扁桃体切除术缓解该病症状的疗效。
本研究纳入16例腺样体扁桃体切除术后出现舌扁桃体肥大的儿童。采用计算机断层扫描(CT)和/或磁共振成像(MRI)检测病变。他们接受了舌扁桃体切除术,并给予特殊的麻醉护理,术前和术后均进行了纤维喉镜检查和多导睡眠图监测。对患者进行了至少1年的随访。
3例出现术后气道梗阻,由舌根水肿引起。10例打鼾和呼吸暂停症状完全改善。尽管舌扁桃体已完全切除,但仍有6例检测到持续性打鼾,2例检测到呼吸暂停。唐氏综合征、黏多糖贮积症和肥胖可能是症状持续存在的潜在因素。
舌扁桃体肥大可能是腺样体扁桃体切除术后儿童阻塞性睡眠呼吸暂停的病因,舌扁桃体切除术对这些病例是一种有效的治疗方法,然而围手术期气道问题应予以预期并能安全处理。舌扁桃体切除术后症状持续可能归因于并存疾病,如颅面畸形、肥胖和/或黏多糖贮积症。