Dietz W H, Bandini L G, Gortmaker S
New England Medical Center, Boston.
Klin Padiatr. 1990 Mar-Apr;202(2):69-72. doi: 10.1055/s-2007-1025490.
The data reviewed here emphasize the suggestion that obesity in children is the result of an interaction between a susceptible host and an environment that promotes the disease. A variety of environmental factors are related to childhood obesity. Nonetheless, except for television viewing, the behavioral correlates of the environmental associations remain unspecified. The goal for the coming decade is the identification of these behaviors, and their modification. The last five years have witnessed a dramatic increase in our understanding of the components of energy expenditure, and their relationship to the development of obesity. Both the early studies of Griffith and Payne, and the more recent studies of infants by Roberts suggest that reduced energy spent on activity may be the constitutional feature that accounts for an increased susceptibility to the disease. Reduced energy spent on activity may also account for some of the epidemiologic correlates. For example, differences in activity may account for the regional, seasonal, and population density effects on obesity, as well as some of the family line variables such as parental obesity, and family size. All represent promising sources for future investigation.
此处回顾的数据强调了这样一种观点，即儿童肥胖是易感宿主与促发该疾病的环境之间相互作用的结果。多种环境因素与儿童肥胖相关。然而，除了看电视外，环境关联的行为相关性仍不明确。未来十年的目标是识别这些行为并加以改变。在过去五年里，我们对能量消耗的组成部分及其与肥胖发展的关系的理解有了显著增加。格里菲思和佩恩早期的研究以及罗伯茨最近对婴儿的研究均表明，用于活动的能量减少可能是导致对该疾病易感性增加的体质特征。用于活动的能量减少也可能解释了一些流行病学相关性。例如，活动差异可能解释了肥胖的区域、季节和人口密度效应，以及一些家族谱系变量，如父母肥胖和家庭规模。所有这些都是未来研究的有希望的来源。
