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脾动脉盗血综合征病例报告:通过脾动脉解剖变异展示门静脉高血流机制及其与多普勒速率的相关性

Case report of splenic artery steal syndrome: demonstration of portal hyperflow mechanism by anatomic variant of the splenic artery and correlation with Doppler rates.

作者信息

Pérez J, Grande L G, Achécar L, Moreno A, Fortún J, Arranz G, Tejedor M, Blázquez J, Sánchez J, Moreno A, Pian H, Bárcena R

机构信息

Department of Gastroenterology, Hospital Ramón y Cajal, Madrid, Spain.

出版信息

Transplant Proc. 2011 Jul-Aug;43(6):2269-71. doi: 10.1016/j.transproceed.2011.05.022.

DOI:10.1016/j.transproceed.2011.05.022
PMID:21839253
Abstract

We report the case of a liver transplant recipient who developed a "splenic artery steal syndrome" (SASS) successfully treated by partial splenic embolization (PSE). Interestingly, because the patient presented an anatomic variant of the splenic artery (SA) originating from the superior mesenteric artery (SMA), improvement was observed in hepatic artery (HA) flow following PSE that could only be explained by decreased portal perfusion and not by the derivation from the SA.

摘要

我们报告了一例肝移植受者发生“脾动脉盗血综合征”(SASS),经部分脾栓塞术(PSE)成功治疗的病例。有趣的是,由于该患者存在脾动脉(SA)起源于肠系膜上动脉(SMA)的解剖变异,PSE术后肝动脉(HA)血流有所改善,这只能通过门静脉灌注减少来解释,而不能通过SA的血流分流来解释。

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