Roe D A
Division of Nutritional Sciences, Cornell University, Ithaca, New York.
Clin Geriatr Med. 1990 May;6(2):319-34.
The aging process alters body composition so that nutritional status changes as we get older. The aging process shows interindividual variability in its rate of development. Determinants of the rates of aging of systems and tissues are largely genetic. Premature aging of cells and tissues is due to genetic factors and to long-term exposure to physical or chemical environments that cause irreversible tissue damage. Whereas maximal lifespan is fixed for us all, individuals vary in life expectancy both because of variability in the risk of genetic disease which shortens life and because of variable capability for avoidance of those factors in our environment which cause early aging. Early aging as well as geriatric disease foreshorten life, but both can be prevented to some extent by diet or by diet and exercise. Diseases that can be nutritionally prevented, giving us a greater chance of achieving our genetically determined lifespans, include nutritional deficiency states and chronic diet-related diseases such as non-insulin-dependent diabetes, hypertension, coronary artery disease, and cancer. Disabilities resulting from these diseases and from degenerative arthritis are also subject to modulation by diet. The nutritional requirements of the elderly are mostly similar to those of younger people. Elderly usually need fewer calories and similar nutrient intakes compared with those of younger people. Elderly with higher needs for specific nutrients include homebound or institutionalized people who lack sunlight exposure and therefore require more vitamin D. Nutritional requirements to promote longer life expectancy and freedom from disabilities that result from chronic disease include restriction of food energy and fat. Nutritional assessment of the elderly is aimed at identifying not only the presence of deficiency states but also states of nutrient excess and chronic diet-related diseases. There are certain problems in carrying out nutritional assessment in the elderly, but techniques are now available which make valid assessment possible even in the oldest old. Those who live longest have less genetic risk of premature aging, but as a result of native intelligence, education, coping skills, and higher socioeconomic status, they also have a greater likelihood of eating a diet that best meets their long-term nutritional needs. Those most at risk for developing malnutrition as they get older are those who lack food access because of poverty, because of disability resulting from chronic geriatric disease, or because of a combination of these factors. Malnutrition is found in elderly in our society who live in their own homes if they are indigent, isolated, and homebound because of disability.(ABSTRACT TRUNCATED AT 400 WORDS)
衰老过程会改变身体组成,因此随着年龄增长,营养状况也会发生变化。衰老过程在其发展速度上存在个体差异。系统和组织的衰老速度的决定因素很大程度上是遗传因素。细胞和组织的过早衰老归因于遗传因素以及长期暴露于导致不可逆组织损伤的物理或化学环境。虽然我们所有人的最大寿命是固定的,但个体的预期寿命各不相同,这既是因为导致寿命缩短的遗传疾病风险存在差异,也是因为避免环境中那些导致过早衰老因素的能力不同。过早衰老以及老年疾病会缩短寿命,但通过饮食或饮食与运动在一定程度上都可以预防。可以通过营养预防从而让我们更有可能达到基因决定寿命的疾病,包括营养缺乏状态以及慢性饮食相关疾病,如非胰岛素依赖型糖尿病、高血压、冠状动脉疾病和癌症。这些疾病以及退行性关节炎导致的残疾也可通过饮食进行调节。老年人的营养需求大多与年轻人相似。与年轻人相比,老年人通常需要的热量更少,营养摄入量相似。对特定营养素需求较高的老年人包括居家或住在养老院且缺乏阳光照射因而需要更多维生素D的人。促进更长预期寿命以及避免慢性疾病导致残疾的营养需求包括限制食物能量和脂肪。对老年人的营养评估不仅旨在确定是否存在缺乏状态,还包括营养素过量状态和慢性饮食相关疾病。对老年人进行营养评估存在某些问题,但现在有一些技术,即使对最年长的老年人也能进行有效的评估。寿命最长的人过早衰老的遗传风险较低,但由于天生的智力、教育、应对技能以及较高的社会经济地位,他们也更有可能食用最能满足其长期营养需求的饮食。随着年龄增长最容易出现营养不良风险的人是那些因贫困、慢性老年疾病导致残疾或这些因素共同作用而无法获得食物的人。在我们社会中,那些因贫困、残疾而居家且孤立无援的老年人,如果住在自己家里也会出现营养不良。(摘要截选至400字)