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Killing of Mycobacterium avium: insights provided by the use of recombinant cytokines.

作者信息

Bermudez L E, Young L S

机构信息

Kuzell Institute for Arthritis and Infectious Diseases, Pacific Presbyterian Medical Center, San Francisco, CA.

出版信息

Res Microbiol. 1990 Feb;141(2):241-3. doi: 10.1016/0923-2508(90)90037-q.

DOI:10.1016/0923-2508(90)90037-q
PMID:2189170
Abstract
摘要

相似文献

1
Killing of Mycobacterium avium: insights provided by the use of recombinant cytokines.
Res Microbiol. 1990 Feb;141(2):241-3. doi: 10.1016/0923-2508(90)90037-q.
2
Intracellular killing of mycobacteria.分枝杆菌的细胞内杀伤作用。
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3
Production of transforming growth factor-beta by Mycobacterium avium-infected human macrophages is associated with unresponsiveness to IFN-gamma.鸟分枝杆菌感染的人类巨噬细胞产生转化生长因子-β与对γ干扰素无反应有关。
J Immunol. 1993 Mar 1;150(5):1838-45.
4
Interleukin-6 antagonizes tumor necrosis factor-mediated mycobacteriostatic and mycobactericidal activities in macrophages.白细胞介素-6可拮抗肿瘤坏死因子介导的巨噬细胞对分枝杆菌的抑菌和杀菌活性。
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Induction of IL-1 beta, IL-6, TNF-alpha, GM-CSF and G-CSF in human macrophages by smooth transparent and smooth opaque colonial variants of Mycobacterium avium.鸟分枝杆菌光滑透明和光滑不透明菌落变体对人巨噬细胞中白细胞介素-1β、白细胞介素-6、肿瘤坏死因子-α、粒细胞-巨噬细胞集落刺激因子和粒细胞集落刺激因子的诱导作用。
J Med Microbiol. 1994 Feb;40(2):129-33. doi: 10.1099/00222615-40-2-129.
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Recombinant granulocyte-macrophage colony-stimulating factor (GM-CSF) or tumour necrosis factor-alpha (TNF-alpha) activate human alveolar macrophages to inhibit growth of Mycobacterium avium complex.重组粒细胞-巨噬细胞集落刺激因子（GM-CSF）或肿瘤坏死因子-α（TNF-α）可激活人肺泡巨噬细胞，以抑制鸟分枝杆菌复合体的生长。
Clin Exp Immunol. 1994 Oct;98(1):169-73. doi: 10.1111/j.1365-2249.1994.tb06625.x.
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Recombinant granulocyte-macrophage colony-stimulating factor activates human macrophages to inhibit growth or kill Mycobacterium avium complex.重组粒细胞-巨噬细胞集落刺激因子激活人类巨噬细胞以抑制鸟分枝杆菌复合体生长或将其杀死。
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Killing intracellular mycobacteria in in vitro macrophage systems: what may be the role of known host microbicidal mechanisms?在体外巨噬细胞系统中杀灭细胞内分枝杆菌：已知宿主杀菌机制可能发挥什么作用？
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Pentoxifylline impairs macrophage defense against Mycobacterium avium complex.
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Late acquisition of hyporesponsiveness to lipopolysaccharide by Mycobacterium avium-infected human macrophages in producing tumor necrosis factor-alpha but not interleukin-1 beta and -6.鸟分枝杆菌感染的人类巨噬细胞在产生肿瘤坏死因子-α而非白细胞介素-1β和-6方面对脂多糖的低反应性的后期获得。
J Infect Dis. 1996 Apr;173(4):1030-4. doi: 10.1093/infdis/173.4.1030.

引用本文的文献

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Mycobacterium avium Complex Extracellular Vesicles Attenuate Inflammation via Inducing IL-10.鸟分枝杆菌复合群细胞外囊泡通过诱导白细胞介素-10减轻炎症。
Int J Mol Cell Med. 2018 Fall;7(4):241-250. doi: 10.22088/IJMCM.BUMS.7.4.241. Epub 2019 Apr 7.
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Mycobacterium avium biofilm attenuates mononuclear phagocyte function by triggering hyperstimulation and apoptosis during early infection.鸟分枝杆菌生物膜通过在早期感染期间触发过度刺激和细胞凋亡来减弱单核吞噬细胞的功能。
Infect Immun. 2014 Jan;82(1):405-12. doi: 10.1128/IAI.00820-13. Epub 2013 Nov 4.
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Tuberculosis in Birds: Insights into the Mycobacterium avium Infections.
鸟类结核病：对鸟分枝杆菌感染的见解
Vet Med Int. 2011;2011:712369. doi: 10.4061/2011/712369. Epub 2011 Jul 4.
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The modulating effects of proinflammatory cytokines interferon-gamma (IFN-gamma) and tumour necrosis factor-alpha (TNF-alpha), and immunoregulating cytokines IL-10 and transforming growth factor-beta (TGF-beta), on anti-microbial activity of murine peritoneal macrophages against Mycobacterium avium-intracellulare complex.促炎细胞因子γ干扰素（IFN-γ）和肿瘤坏死因子-α（TNF-α），以及免疫调节细胞因子白细胞介素-10（IL-10）和转化生长因子-β（TGF-β）对小鼠腹膜巨噬细胞抗鸟分枝杆菌-胞内分枝杆菌复合体抗菌活性的调节作用。
Clin Exp Immunol. 1999 Mar;115(3):435-42. doi: 10.1046/j.1365-2249.1999.00838.x.
5
Differential potentiation of anti-mycobacterial activity and reactive nitrogen intermediate-producing ability of murine peritoneal macrophages activated by interferon-gamma (IFN-gamma) and tumour necrosis factor-alpha (TNF-alpha).γ干扰素（IFN-γ）和肿瘤坏死因子-α（TNF-α）激活的小鼠腹腔巨噬细胞的抗分枝杆菌活性及产生反应性氮中间产物能力的差异增强作用
Clin Exp Immunol. 1998 Apr;112(1):63-8. doi: 10.1046/j.1365-2249.1998.00554.x.
6
Infection with Mycobacterium avium induces production of interleukin-10 (IL-10), and administration of anti-IL-10 antibody is associated with enhanced resistance to infection in mice.鸟分枝杆菌感染可诱导白细胞介素-10（IL-10）的产生，给小鼠注射抗IL-10抗体与增强其抗感染能力有关。
Infect Immun. 1993 Jul;61(7):3093-7. doi: 10.1128/iai.61.7.3093-3097.1993.
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The Mycobacterium avium complex.鸟分枝杆菌复合体
Clin Microbiol Rev. 1993 Jul;6(3):266-310. doi: 10.1128/CMR.6.3.266.
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Comparative effects of Mycobacterium avium glycopeptidolipid and lipopeptide fragment on the function and ultrastructure of mononuclear cells.鸟分枝杆菌糖脂肽和脂肽片段对单核细胞功能及超微结构的比较作用
Clin Exp Immunol. 1993 Jul;93(1):72-9. doi: 10.1111/j.1365-2249.1993.tb06499.x.
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Response to stimulation with recombinant cytokines and synthesis of cytokines by murine intestinal macrophages infected with the Mycobacterium avium complex.重组细胞因子刺激的反应以及感染鸟分枝杆菌复合群的小鼠肠道巨噬细胞的细胞因子合成
Infect Immun. 1995 Feb;63(2):528-33. doi: 10.1128/iai.63.2.528-533.1995.
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Immunobiology of Mycobacterium avium infection.鸟分枝杆菌感染的免疫生物学
Eur J Clin Microbiol Infect Dis. 1994 Nov;13(11):1000-6. doi: 10.1007/BF02111501.