蛋白激酶的分子药理学 - Suppr

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超能文献

Molecular pharmacology of protein kinases.

作者信息

Hidaka H, Hagiwara M, Chijiwa T

机构信息

Department of Pharmacology, Nagoya University School of Medicine, Japan.

出版信息

Neurochem Res. 1990 Apr;15(4):431-4. doi: 10.1007/BF00969929.

DOI:10.1007/BF00969929

PMID:2201927

Abstract

摘要

相似文献

Molecular pharmacology of protein kinases.蛋白激酶的分子药理学

Neurochem Res. 1990 Apr;15(4):431-4. doi: 10.1007/BF00969929.

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Properties and use of H-series compounds as protein kinase inhibitors.H 系列化合物作为蛋白激酶抑制剂的性质与用途。

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Search for the functional substrate proteins of protein kinases and their selective inhibitors.

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Long-term potentiation: presynaptic enhancement following postsynaptic activation of Ca(++)-dependent protein kinases.

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Protein kinase inhibitors.蛋白激酶抑制剂

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ATP gamma S activates the respiratory burst in electropermeabilized neutrophils: the role of protein kinases.ATPγS激活电通透中性粒细胞中的呼吸爆发：蛋白激酶的作用

Biochem Soc Trans. 1991 Apr;19(2):77S. doi: 10.1042/bst019077s.

Identifying and localizing protein kinases necessary for LTP.

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Peptide inhibitors of PKC and CaMK block induction but not expression of long-term potentiation.

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[Molecular pharmacology on protein kinase C].

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引用本文的文献

PKA and PKC are required for long-term but not short-term in vivo operant memory in Aplysia.PKA 和 PKC 在海兔体内的长时而非短时操作性记忆中是必需的。

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Epac1 mediates protein kinase A-independent mechanism of forskolin-activated intestinal chloride secretion.Epac1介导福斯高林激活的肠氯化物分泌的不依赖蛋白激酶A的机制。

J Gen Physiol. 2010 Jan;135(1):43-58. doi: 10.1085/jgp.200910339.

PKC differentially translocates during spaced and massed training in Aplysia.蛋白激酶C（PKC）在海兔的间隔训练和集中训练过程中发生不同的转位。

J Neurosci. 2009 Aug 19;29(33):10281-6. doi: 10.1523/JNEUROSCI.1533-09.2009.

M1 muscarinic receptor-induced facilitation of ACh and noradrenaline release in the rat bladder is mediated by protein kinase C.毒蕈碱M1受体诱导大鼠膀胱中乙酰胆碱和去甲肾上腺素释放的促进作用由蛋白激酶C介导。

J Physiol. 1996 Oct 1;496 ( Pt 1)(Pt 1):245-54. doi: 10.1113/jphysiol.1996.sp021681.

Ethanol induced alterations in plasma membrane protein phosphorylation of neurons and astrocytes.乙醇诱导神经元和星形胶质细胞质膜蛋白磷酸化的改变。

Mol Cell Biochem. 1994 Jan 12;130(1):41-8. doi: 10.1007/BF01084266.

Protein kinase inhibitors prevent junction dissociation induced by low extracellular calcium in MDCK epithelial cells.蛋白激酶抑制剂可防止低细胞外钙诱导的MDCK上皮细胞连接解离。

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Enhanced synthesis of brain-derived neurotrophic factor in the lesioned peripheral nerve: different mechanisms are responsible for the regulation of BDNF and NGF mRNA.损伤的周围神经中脑源性神经营养因子的合成增强：不同机制负责调节脑源性神经营养因子和神经生长因子的信使核糖核酸。

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本文引用的文献

Isoquinolinesulfonamides, novel and potent inhibitors of cyclic nucleotide dependent protein kinase and protein kinase C.异喹啉磺酰胺，新型强效环核苷酸依赖性蛋白激酶和蛋白激酶C抑制剂。

Biochemistry. 1984 Oct 9;23(21):5036-41. doi: 10.1021/bi00316a032.

An adenosine 3',5'-monophosphate-dependant protein kinase from rabbit skeletal muscle.来自兔骨骼肌的一种3',5'-环磷酸腺苷依赖性蛋白激酶。

J Biol Chem. 1968 Jul 10;243(13):3763-5.

Tissue-specific expression of three distinct types of rabbit protein kinase C.三种不同类型兔蛋白激酶C的组织特异性表达。

Nature. 1987;325(7000):161-6. doi: 10.1038/325161a0.

An inhibitor of protein kinase C, 1-(5-isoquinolinylsulfonyl)-2-methylpiperazine(H-7) inhibits TPA-induced reduction of vincristine uptake from P388 murine leukemic cell.

Leuk Res. 1986;10(9):1063-9. doi: 10.1016/0145-2126(86)90050-0.

Specific binding of a novel compound, N-[2-(methylamino)ethyl]-5-isoquinolinesulfonamide (H-8) to the active site of cAMP-dependent protein kinase.一种新型化合物N-[2-(甲氨基)乙基]-5-异喹啉磺酰胺（H-8）与环磷酸腺苷（cAMP）依赖性蛋白激酶活性位点的特异性结合。

Mol Pharmacol. 1987 May;31(5):523-8.

A fluorescent protein kinase C inhibitor: 1-(1-hydroxy-5-isoquinolinylsulfonyl)piperazine.一种荧光蛋白激酶C抑制剂：1-(1-羟基-5-异喹啉基磺酰基)哌嗪。

Pharmacology. 1988;36(6):365-70. doi: 10.1159/000138324.

Inhibitory effects of aminoglycosides on renal protein phosphorylation by protein kinase C.氨基糖苷类药物对蛋白激酶C介导的肾蛋白磷酸化的抑制作用。

J Pharmacol Exp Ther. 1988 Jan;244(1):355-60.

Cell type-specific expression of protein kinase C isozymes in the rabbit cerebellum.蛋白激酶C同工酶在兔小脑中的细胞类型特异性表达。

J Biol Chem. 1988 Apr 5;263(10):4523-6.

Differential effects of flavonoids as inhibitors of tyrosine protein kinases and serine/threonine protein kinases.黄酮类化合物作为酪氨酸蛋白激酶和丝氨酸/苏氨酸蛋白激酶抑制剂的差异效应。

Biochem Pharmacol. 1988 Aug 1;37(15):2987-92. doi: 10.1016/0006-2952(88)90286-9.

Selective inhibition of catalytic activity of smooth muscle myosin light chain kinase.

J Biol Chem. 1987 Jun 5;262(16):7796-801.

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