[感染性-过敏性心肌炎中心肌收缩力损伤的机制]
[Mechanisms of lesions of myocardial contractility in infectious-allergic myocarditis].
作者信息
Paleev N R, Odinokov V A, Gurevich M A, Smirnov V B
出版信息
Sov Med. 1990(5):22-5.
PMID:2202058
Abstract
Clinical and morphologic analysis of 63 biopsy specimens and of 23 autopsy specimens has shown the leading role of immune inflammation with the development of delayed and immediate type hypersensitivity in injury to myocardial contractility. It is the immune inflammation that is responsible for disorders in electrolyte metabolism, microcirculation, sympathetic nervous system reactivity.
摘要
对63份活检标本和23份尸检标本的临床及形态学分析表明，免疫炎症在心肌收缩力损伤中起主导作用，伴有迟发型和速发型超敏反应的发生。正是免疫炎症导致了电解质代谢、微循环及交感神经系统反应性的紊乱。
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