Ansons A M, Clearkin L, Bryce I
St Paul's Eye Hospital, Liverpool.
Eye (Lond). 1990;4 ( Pt 3):439-44. doi: 10.1038/eye.1990.55.
Forty patients with retinal vein occlusion were studied to investigate possible mechanisms leading to an acute rise in intraocular pressure. A rise in intraocular pressure on recumbency was found in 64% of eyes with a central retinal vein occlusion and 65% of eyes with a branch retinal vein occlusion. This finding correlated with glucose tolerance test score, and may be the result of abnormal glucose tolerance in these patients. Following provocative testing using pilocarpine and phenylephrine, no eye showed irido-corneal contact or angle closure, however over 50% of eyes release pigment into the anterior chamber. Fifty-two per cent of central retinal vein occlusion and 71% of branch retinal vein occlusion eyes that released pigment showed a rise in intraocular pressure.
对40例视网膜静脉阻塞患者进行了研究，以探讨导致眼压急性升高的可能机制。在64%的视网膜中央静脉阻塞眼和65%的视网膜分支静脉阻塞眼中发现卧位时眼压升高。这一发现与葡萄糖耐量试验评分相关，可能是这些患者葡萄糖耐量异常的结果。在使用毛果芸香碱和去氧肾上腺素进行激发试验后，没有一只眼睛出现虹膜角膜接触或房角关闭，但超过50%的眼睛向前房释放色素。释放色素的视网膜中央静脉阻塞眼中有52%、视网膜分支静脉阻塞眼中有71%眼压升高。
