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Evidence for noradrenergic involvement in episodic prolactin and growth hormone release in ovariectomized rats.

作者信息

Negro-Vilar A, Ojeda S R, Advis J P, McCann S M

出版信息

Endocrinology. 1979 Jul;105(1):86-91. doi: 10.1210/endo-105-1-86.

DOI:10.1210/endo-105-1-86
PMID:221208
Abstract
摘要

相似文献

1
Evidence for noradrenergic involvement in episodic prolactin and growth hormone release in ovariectomized rats.去卵巢大鼠发作性催乳素和生长激素释放中去甲肾上腺素能参与的证据。
Endocrinology. 1979 Jul;105(1):86-91. doi: 10.1210/endo-105-1-86.
2
Evidence for alpha-adrenergic regulation of episodic growth hormone and prolactin secretion in the undisturbed male rat.未受干扰的雄性大鼠中α-肾上腺素能调节间歇性生长激素和催乳素分泌的证据。
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3
Differential effects of pharmacological manipulations of central alpha 1- and alpha 2-adrenergic receptors on the secretion of thyrotropin and growth hormone in male rats.中枢α1和α2肾上腺素能受体的药理学调控对雄性大鼠促甲状腺激素和生长激素分泌的不同影响。
Endocrinology. 1982 Mar;110(3):796-804. doi: 10.1210/endo-110-3-796.
4
Evidence for a role of alpha-adrenergic mechanisms in regulation of episodic growth hormone secretion in the rat.α-肾上腺素能机制在调节大鼠间歇性生长激素分泌中作用的证据。
Endocrinology. 1977 Mar;100(3):722-8. doi: 10.1210/endo-100-3-722.
5
Further characterization of putative alpha--adrenergic receptors in brain that affect blood pressure and the secretion of ACTH, GH and renin in dogs.对犬脑中影响血压以及促肾上腺皮质激素、生长激素和肾素分泌的假定α-肾上腺素能受体的进一步表征。
Endocrinol Exp. 1982 Nov;16(3-4):191-205.
6
Extreme elevations in plasma norepinephrine associated with decreased alpha-adrenergic responsiveness in major depressive disorder: two case reports.重度抑郁症患者血浆去甲肾上腺素极度升高与α-肾上腺素能反应性降低相关：两例病例报告
J Clin Psychopharmacol. 1983 Feb;3(1):39-41.
7
Catecholamine synthesis inhibitors acutely modulate [3H]estradiol binding by specific brain areas and pituitary in ovariectomized rats.儿茶酚胺合成抑制剂可急性调节去卵巢大鼠特定脑区和垂体的[3H]雌二醇结合。
Endocrinology. 1983 Sep;113(3):855-65. doi: 10.1210/endo-113-3-855.
8
The effects of estradiol on prolactin and growth hormone secretion in cultured pituitary cells from intact and ovariectomized rats.雌二醇对完整和去卵巢大鼠垂体培养细胞中催乳素和生长激素分泌的影响。
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Evidence for the involvement of central epinephrine systems in the regulation of luteinizing hormone, prolactin, and growth hormone release in female rats.
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Radioligand binding studies on hypothalamic noradrenergic receptors during the estrous cycle or after steroid injection in ovariectomized rats.在动情周期或对去卵巢大鼠注射类固醇后，对下丘脑去甲肾上腺素能受体进行放射性配体结合研究。
Brain Res. 1979 Jun 8;168(3):652-5. doi: 10.1016/0006-8993(79)90323-8.

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Cervical stimulation activates A1 and locus coeruleus neurons that project to the paraventricular nucleus of the hypothalamus.宫颈刺激激活投射到下丘脑室旁核的 A1 和蓝斑神经元。
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Physiological role of alpha-melanocyte-stimulating hormone in modulating the secretion of prolactin and luteinizing hormone in the female rat.α-黑素细胞刺激素在调节雌性大鼠催乳素和促黄体生成素分泌中的生理作用。
Proc Natl Acad Sci U S A. 1984 Dec;81(24):8004-8. doi: 10.1073/pnas.81.24.8004.
3
Up- and down- regulation of central postsynaptic alpha 2 receptors reflected in the growth hormone response to clonidine in reserpine-pretreated rats.
利血平预处理大鼠中，生长激素对可乐定的反应所反映的中枢突触后α2受体的上调和下调。
Psychopharmacology (Berl). 1982;77(4):327-31. doi: 10.1007/BF00432764.
4
Prolactin-lowering and -releasing drugs. Mechanisms of action and therapeutic applications.降低和释放催乳素的药物。作用机制及治疗应用。
Drugs. 1983 Apr;25(4):399-432. doi: 10.2165/00003495-198325040-00004.
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Neuropeptide Y affects secretion of luteinizing hormone and growth hormone in ovariectomized rats.神经肽Y对去卵巢大鼠促黄体生成素和生长激素分泌的影响。
Proc Natl Acad Sci U S A. 1985 Jan;82(2):561-4. doi: 10.1073/pnas.82.2.561.
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Pulsatile peptide secretion: encoding of brain messages regulating endocrine and reproductive functions.脉冲式肽分泌：调节内分泌和生殖功能的脑信号编码。
Environ Health Perspect. 1987 Nov;75:37-43. doi: 10.1289/ehp.877537.
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Effects of sex steroids on growth hormone responses to clonidine and GHRH in reserpine pretreated rats.性类固醇对利血平预处理大鼠生长激素对可乐定和生长激素释放激素反应的影响。
J Neural Transm. 1988;71(2):99-113. doi: 10.1007/BF01245252.
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Serotonergic influence on the growth hormone response to clonidine in rat.5-羟色胺能对大鼠可乐定生长激素反应的影响。
J Neural Transm. 1987;69(1-2):105-14. doi: 10.1007/BF01244101.